Beninoliveoil

cancer Cancer become any age group person may cancer, but nearly all types are more common in middle aged and elderly people than in young people. Skin is the most common type of cancer for both men and women. The next most common type among men is prostate cancer; among women, it is breast cancer. Lung cancer, however, is the leading cause of death from cancer for both men and women . Brain cancer and leukemia are the most common cancers in children and young adults.

The more we can learn about what causes cancer, the more likely we are to find ways to prevent it. Scientists study patterns of cancer in the population to look for factors that affect the risk of developing this disease. In the laboratory, they explore possible causes of cancer and try to determine what actually happens when normal cells become cancerous.

Our current understanding of the causes of cancer is incomplete, but it is clear that cancer is not caused by an injury, such as a bump or bruise. And although being infected with certain viruses may increase the risk of some types of cancer, cancer is not contagious no one can “catch” cancer from another person.

Cancer develops gradually as a result of a complex mix of factors related to environment, lifestyle, and heredity. Scientists have identified many risk factors that increase the chance of getting cancer. They estimate that about 80 percent of all cancers are related to the use of tobacco products, to what we eat and drink, or, to a lesser extent, to exposure to radiation or cancer-causing agents (carcinogens) in the environment and the workplace. Some people are more sensitive than others to factors that can cause cancer.

Many risk factors can be avoided. Others, such as inherited risk factors, are, unavoidable. It is helpful to be aware of them, but it Is also important to keep in mind that not everyone with a particular risk factor for cancer actually gets the disease; in fact, most do not. People at risk can help protect themselves by avoiding risk factors where possible and by getting regular checkups so that, if cancer develops, it is likely to be found early.

These are some of the factors that are known to increase the risk of cancer.

– Tobacco causes cancer. In fact, smoking tobacco, using “smokeless” tobacco, and being regularly exposed to environmental tobacco smoke without smoking are responsible for one-third of all cancer deaths. Smoking accounts for more than 85 percent of all lung cancer deaths. If you smoke, your risk of getting lung cancer is affected by the number and type of cigarettes you smoke and how long you have been smoking. Overall, for those who smoke one pack a day, the chance of setting lung cancer is about 10 times greater than for nonsmokers.

Smokers are also more likely than nonsmokers to develop several other types of cancer (such as oral cancer and cancers of the larynx, esophagus, pancreas, bladder, kidney, and cervix). The risk of cancer begins to decrease when a smoker quits, and the risk continues to decline gradually each year after quitting.

The use of smokeless tobacco (chewing, tobacco and snuff) causes cancer of the mouth and throat. Pre-cancerous conditions, or tissue changes that may lead to cancer, begin to go away after a person stops using smokeless tobacco.

Exposure to environmental tobacco smokes, also called involuntary smoking, increases the risk of lung cancer for nonsmokers. The risk goes up 30 percent or more for a nonsmoking spouse of a person who smokes. Involuntary smoking causes about 4,000 lung cancer deaths in this country each year.

If you use tobacco in any form and you need help quitting, talk with your doctor or dentist, or join a smoking cessation group sponsored by a local hospital or voluntary organization.

Your choice of foods may affect your chance of developing cancer. Evidence points to a link between a high-fat diet and certain cancers, such as cancer of the breast, colon, uterus, and prostate. Being seriously overweight appears to be linked to increased rates of cancer of the prostate, pancreas, uterus, Colon, and ovary, and to breast cancer in older women. On the other hand, studies suggest that foods containing fiber and certain nutrients help protect us against some types of cancer. You may be able to reduce your cancer risk by making some simple food choices. Try to have a varied, well-balanced diet that includes generous amounts of foods that are high in fiber, vitamins, and minerals. At the same time, try to cut down on fatty foods. You should eat five servings of fruits and vegetables each day, choose more whole-grain breads and cereals, and cut down on eggs, high-fat meat, high-fat dairy products (such as whole milk, butter, and most cheeses), salad dressings, margarine, and cooking oils.
Ultraviolet radiation from the sun and from other sources (such as sunlamps and tanning booths) damages the skin and can cause skin cancer. (Two types of ultraviolet radiation–UVA and UVB–are explained in the Medical Terms section.) Repeated exposure to ultraviolet radiation increases the risk of skin cancer, especially if you have fair skin or freckle easily. The sun is ultraviolet rays are strongest during the summer from about 11 a.m. to about 3 p.m. (daylight saving time). The risk is greatest at this time, when the sun is high overhead and shadows are short. As a rule, it is best to avoid the sun when your shadow is shorter than you are.

Protective clothing, such as a hat and, long sleeves, can help block the sun’s harmful rays. You can also use sunscreens to help protect yourself. Sunscreens are rated in strength according to their SPF (sun protection factor), which ranges from 2 to 30 and higher. Those rated 15 to 30 block most of the sun’s harmful rays.

Drinking, large amount of alcohol increases the risk of cancer of the mouth, throat, esophagus, and larynx. (People who smoke cigarettes and drink alcohol have an especially high risk of getting these cancers.) Alcohol can damage the liver and increase the risk of liver cancer. Some studies suggest that drinking alcohol also increases the risk of breast cancer. So if you drink at all, do so in moderation-not more than one or two drinks a day.
X-rays used for diagnosis expose you to very little radiation and the benefits nearly always outweigh the risks. However, repeated exposure can be harmful, so it is a good idea to talk with your doctor or dentist about the need for each x-ray and ask about the use of shields to protect other parts of your body. Before 1950, X-rays were used to treat non-cancerous conditions (such as an enlarged thymus, enlarged tonsils and adenoids, ringworm of the scalp, and acne) in children and young adults. People who have received radiation to the head and neck have a higher-than-average risk of developing thyroid cancer years later. People with a history of such treatments should report it to their doctor and should have a careful exam of the neck every 1 or 2 years.

Also, radiation used in the treatment of some types of cancer can increase the risk of developing a second cancer. Patients having radiation therapy may want to discuss this issue with their doctor.

Chemicals and other substances in the workplace being exposed to substances such as metals, dust chemicals, or pesticides at work can increase the risk of cancer. Asbestos, nickel, cadmium, uranium, radon, vinyl chloride, benzidene, and benzene are well-known examples of carcinogens in the workplace. These may act alone or along with another carcinogen, such as cigarette smoke. For example, inhaling asbestos fibers increases the risk of lung diseases, including cancer, and the cancer risk is especially high for asbestos workers who smoke. It is important to follow work and safety rules to avoid contact with dangerous materials.

: Many women use estrogen therapy to control the hot flashes, vaginal dryness, and osteoporosis (thinning of the bones) that may occur during menopause. However, studies show that estrogen use increases the risk of cancer of the uterus. Other studies suggest an increased risk of breast cancer among women who have used high doses of estrogen or have used estrogen for a long time. At the same time, taking estrogen may reduce the risk of heart disease and osteoporosis.

The risk of uterine cancer appears to be less when progesterone is used with estrogen than when estrogen is used alone. But some scientists are concerned that the addition of progesterone may also increase the CANCER risk of breast cancer.

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Out of ten person, one person suffering fron gestrities and out of them hundred one person suffering from stoch cancer.If the cancer can be identified at a time when it only involves the superficial lining layer of the stomach and only involves a few cells and such a stomach cancer is surgically removed 90 % of these individuals are likely to have a normal life span . However if the stomach cancer has involved all the layers of the stomach this chance decreases to 50% and if it has spread outside the stomach or to distant organs of the body the chances of surviving in the long term are extremely gloomy .

1. Benign stomach (gastric ) ulcer , in most instances these benign ulcers in the stomach do not become malignant although in a few cancerous change can supervene after many years . Helicobacter pylori infection of the stomach appears to be an important cause of gastric ulcers .

2. Chronic duodenal ulcer , this is a common condition of the duodenum , the organ situated next to the stomach . we can reassure those with this condition that even after many years of having a chronic duodenal ulcer , the chances of it becoming malignant are almost nil.

3. Gastritis with low or absent stomach acid production . This situation can occur in an uncommon illness. Pernicious anemia and it can also occur without any obvious reason . Here absent or low levels of stomach acid which is normally produced to aid digestion is associated with the disappearance of gastric glands and this is called atrophic gastritis . Atrophic gastritis is a pre malignant condition .

Inherited causes :family history of stomach cancer ,such a history in a near relative doubles a person’s chances of developing stomach cancer during their life time

Type A blood group ,the common blood groups are A,B,O ,and AB. A person with type A blood has a 20% increased chance of developing stomach cancer during their life time compared to those with other blood groups

Personal health risks: presence of Helicobacter pylori in the stomach .this is an organism which in recent has been associated not only with stomach cancer but also with gastric and duodenal ulcers and with inflammatory changes called gastritis . why Helicobacter infection is present in some individuals but not in others is at present in some individuals but not in others is at present is uncertain , although it may be linked to both undesirable Helicobacter infection probably cause no symptoms . Helicobacter Pylori infection can usually be eradicated by the use of suitable antibiotics . It has also been suggested recently by Australian scientists that the regular intake of acidophilus bacteria (found in some brands of yoghurt and in some bacteria supplements )can also eradicate Helicobacter infection. Previous surgical removal of part of the stomach (partial gastrectomy ) for a non malignant condition , such as a gastric or duodenal ulcer . This become a stomach cancer risk some 15-40 years after the stomach has been removed . Pernicious anemia ,Individuals with this uncommon condition not only have anemia but also have no normal stomach acid production . They develop the condition previously described , atrophic gastritis . Individuals with atrophic gastritis have a 10 %chance of developing stomach cancer during their life time . Low or absent stomach acid production with atrophic gastritis , even in the absence of pernicious anemia , this is a risk for the future development of stomach cancer

Life style health risks : Dietary factors, a special risk for stomach cancer is a diet which is low in vegetables , fruit and cereals and particularly those fruits , vegetables and cereals which contain a lot of beta-carotene , vitamin-c and vitamin-e

A diet high in pickled . smoked , salted or cured food or foods preserved with nitrate , such as salami ,sausages , hot dogs , smoked meat , smoked fish or pickled food of any kind are also risks for stomach cancer . These dietary factors are probably important because the risk foods described above all seem to produce carcinogens

called nitrosamines . Also vitamin-C acts as antioxidant and has other actions which neutralize the effects of nitrosamines . Dietary factors are probably the most important single cause of stomach cancer . Smoking , a recent Australian study which examined all the scientific evidence published over the years has found that smoking is likely to be an important contributory cause of stomach cancer .

1.Dietary changes it is particularly important to have a high consumption of fruit ,vegetables and cereals which contain beta-carotene ,vitamin-c and vitamin-e and at the same time avoid or eat very little pickled ,smoked ,salted ,cured and nitrate –preserved foods .In a recently reported study from China ,where stomach cancer is still relatively common ,the daily use of vitamin-e ,beta carotene and selenium supplements decreased stomach cancer risk by one-fifth .

2. Avoid smoking ,hints and guidance about quitting smoking are described

3. Eradicate Helicobacter Pylori , if helicobacter infection has been shown to be present ,suitable antibiotics can be used recent research suggests that the acidophilus bacteria found in some dietary supplements may also been effective way to eliminate helicobacter infection

4. Aspirin based mainly on experimental data ,the regular use of aspirin as a preventive for stomach cancer has been advanced . However the human evidence of a preventive role for aspirin in stomach cancer is insufficient at present to make such a recommendation .

In alternative medicine in homoeopathy medicine can cure cancer in various age group of person. In early stage of cancer may help to patient life Many people with cancer want to learn all they can about their disease and their treatment choices so they can take an active part in decisions about their medical care. Often, it helps to make a list of questions to ask the doctor. Patients may take notes or, with the doctor’s consent, tape record the discussion. Some patients also find it helps to have a family member or friend with them when they talk with the doctor to take part in the discussion, to take notes, or just to listen.

• Here are some questions may want to ask the doctor: What are the chances that the treatment will be successful?
• Would a clinical trial be appropriate for me?
• What are the risks and possible side effects of each treatment?

Although the side effects of radiation therapy can be unpleasant, the doctor can usually treat or control them. It also helps to know that, in most cases, they are not permanent.
number of white blood cells, cells that help protect the body against infection
—the side effects of chemotherapy depends mainly on the drugs and doses the patient receives. Generally, anticancer drugs affect cells that divide rapidly. These include blood cells, which fight infection, help the blood to clot, or carry oxygen to all parts of the body. When blood cells are affected by anticancer drugs, patients are more likely to get infections, may bruise or bleed easily, and may have less energy. Cells that line the digestive tract also divide rapidly. As a result of chemotherapy, patients may have side effects, such as loss of appetite, nausea and vomiting, hair loss, or mouth sores. For some patients, the doctor may prescribe medicine to help with side effects, especially with nausea and vomiting. Usually, these side effects gradually go away during the recovery period or after treatment stops. Hair loss another side effect of chemotherapy, is a major concern for many patients. Some chemotherapy drugs only cause the hair to thin out, while others may result in the loss of all body hair. Patients may feel better if they decide how to handle hair loss before starting treatment.

In some men and women, chemotherapy drugs cause changes that may result in a loss of fertility (the ability to have children). Loss of fertility may be temporary or permanent depending on the drugs used and the patient’s age. For men, sperm banking before treatment may be a choice. Women’s menstrual periods may stop, and they may have hot flashes and vaginal dryness. Periods are more likely to return in young women. In some cases, bone marrow transplantation and peripheral stem cell support are used to replace tissue that forms blood cells when that tissue has been destroyed by the effects of chemotherapy or radiation therapy.

–Hormone therapy can cause a number of side effects. Patients may have nausea and vomiting, swelling or weight gain, and, in some cases, hot flashes. In women, hormone therapy also may cause interrupted menstrual periods, vaginal dryness, and, sometimes, loss of fertility. Hormone therapy in men may cause impotence, loss of sexual desire, or loss of fertility. These changes may be temporary, long lasting, or permanent.

HOMOEOPATHIC TREATMENT/MEDICINES

CHELIDINIUM : It has also cured cancer of the stomach when in a ,vomiting for cancer of throat , mouth or stomach .

Condurango : In cancer of oesophagus or stomach . There are many other medicines , only few have been mentioned

When Virginia Livingston was a student at Bellevue Medical College her pathology teacher mentioned, rather disparagingly, that there was a woman pathologist at Cornell who thought Hodgkin’s disease (a form of glandular cancer) was caused by avian tuberculosis [1]. This lady had published, but no one had confirmed her findings. Afterwards, Livingston compared slides of both. In Hodgkin’s, the large multinucleated giant cells were called Reed–Sternberg cells. They were similar to the giant cells of tuberculosis, which formed to engulf the tubercle bacilli. Livingston stored away in her memory that this lady pathologist was probably right but she would have a difficult time in gaining acceptance.

 By 1931, Pathologist Elsie L’Esperance was seeing ‘acid fast’ tuberculosis-like bacteria riddling her Hodgkin’s cancer tissue samples. And that germ, once injected into guinea pigs, caused them to come down with Hodgkin’s too, fulfilling Koch’s postulates. L’Esperance brought her stained slides to former teacher and prominent Cornell cancer pathologist James Ewing. Ewing initially confirmed that her tissue slides were indeed Hodgkin’s. But when he found out that her slides came through guinea pig inoculation of the avian (fowl) tuberculosis she had found in humans with Hodgkin’s, Ewing, visibly upset, said that the slides then could not be cancer.

It betrayed his checkered history of high-placed medical politician. In 1907, you could have approached Dr. James Ewing about a cancer germ, and he would have embraced you over it. At that time, both for he and the rest of the nations medical authorities, it was not a question of whether cancer was caused by a germ, but which one. Was not it Ewing, at one time, who had proclaimed that tuberculosis followed Hodgkin’s cancer “like a shadow”?

But shortly after, James Ewing, “the Father of Oncology”, sent a sword thru the heart of an infectious cause of cancer with “Neoplastic Diseases” [2], becoming an ambitious zealot for radiation therapy with the directorship of what would one day be called Sloan–Kettering squarely on his mind. His entry lay in prominent philanthropist James Douglas. A vote for Ewing, Douglas knew, was a vote for continued radiation and James Douglas began sizeable uranium extraction operations from Colorado mines thru his company, Phelps Dodge, Inc.[34].

Soon Sloan became known as a radium hospital and went from an institution with a census of less than 15% cancer patients, separated by partition, lest their disease spread to others, to a veritable cancer center. But the very history of radiation revealed its flaws, and by the early 1900s nearly 100 cases of leukemia were documented in radium recipients and not long thereafter it was determined that approximately 100 radiologists had contracted that cancer in the same way [3].

Still, Ewing, by now an Honorary Member of the American Radium Society, persisted.

Elise L’Esperance was anything but alone in linking Hodgkin’s to a germ called Avium or fowl tuberculosis. Historically Sternberg himself, discoverer of Hodgkin’s trade-mark Reed–Sternberg cell, believed Hodgkin’s was caused by tuberculosis. Both Fraenkel and Much [35] held, as L’Esperance, that it was caused by a peculiar form of tuberculosis, such as Avium or Fowl tuberculosis, and of all the cancers, debate over the infectious cause of Hodgkin’s waxed the hottest.

Into this arena L’Esperance stepped in 1931, with few listening. She would publish Studies in Hodgkin’s Diseases [4] in an issue of Annals of Surgery. It proved to be the one legacy that no one, not even Ewing, who would soon die from a self-diagnosed cancer, could take away.

 ”Our (cancer) cultures were scrutinized over and over again. Strains were sent to many laboratoriesfor identification. None could really classify them. They were something unknown. They had many forms but they always grew up again to be the same thing no matter how they were cultured. They resembled the mycobacteria more than anything else. The tubercle bacillus is a mycobacterium or fungoid bacillus.”–Virginia Livingston, 1972

Virginia Wuerthele-Caspe Livingston was born in Meadville, Pennsylvania and went on to obtain impeccable credentials. Graduating from Vassar, she received her M.D. from N.Y.U. The first female medical resident ever in New York City, with time Livingston became a Newark school physician where one day a staff nurse asked medical assistance.

Already diagnosed with Reynaud’s syndrome, the tips of this nurses fingers were ulcerated and bled intermittently. Livingston diagnosed Scleroderma. But upon further examination there was a hole in the nasal septa, something that Livingston had previous seen in the mycobacterial diseases TB and Leprosy.

So Livingston approached dermatologist Eva Brodkin and a pathologist for confirmation, all the while convinced that mycobacterial infection was causing the Scleroderma. She then preformed cultures from a sterile nasal swab – mycobacteria appeared, everywhere [1]. Injected into experimental chicks and guinea pigs, all but a couple died. Upon autopsy, the guinea pigs had indeed developed the hardened skin patches of Scleroderma. . . some of which were cancerous.

Livingston, now possessed, solicited fresh sterile specimens of cancer from any operating room that would give them to her. All cancer tissues yielded the same acid-fast mycobacteria. New Jersey Pathologist Roy Allen confirmed her findings. Livingston and Allen then found that they could actually differentiate malignant from benign tissue by their mycobacterial content [5]. But still the explanation for why the cancer germ showed so many different forms was elusive.

Try as she might, part of Virginia Livingston’s problems in an American validation of her multi-shaped cancer germ lay firmly entrenched in the history of medicine, especially in the constantly changing field of microbiology. Louis Pasteur could handle being quickly rushed off a Paris Academy of Sciences podium to escape harsh reaction to his suggestion that children’s milk be boiled first, but he could not tolerate his rival Pierre Bechamp’s statement that a single bacteria could assume many, many forms. On his deathbed, Pasteur was said to have changed his mind when he said: “The terrain is everything”, meaning the culture or milieu that bacteria grew on or in could change their shape or characteristics. But it was too late and even today, most conventional microbiologists deny the existence of such form changing (or pleomorphic) germs.

Robert Koch, Father of Bacteriology and discoverer of tuberculosis, could have helped. When he first worked with the bacteria anthrax, he noticed that anthrax’s classical rod shape became thread-like inside the blood of laboratory mice. And then, after multiplying, they changed again, into the same assumed spore-like forms he later documented in tuberculosis as well.

Aware of what she faced, yet undismayed   Livingston methodically went about proving cancers true cause. First in her line of attack were the long suspected and well-publicized tumor agents of Rous, Bittner and Shope. By photomicrographs, Livingston and her group demonstrated acid-fast mycobacterial forms in each of these so-called “viral” cancers. This included the famed Rous chicken sarcoma.

Early on, Virginia Livingston had decided that she needed help in validating her cancer germ and nobody knew the shapes and staining capacities of mycobacterial-related germs better than Dr. Eleanor Alexander-Jackson of Cornell. As far back as 1928, Eleanor Alexander-Jackson, bacteriologist, had discovered unusual and to that point unrecognized forms of the TB bacillus, including its filterable forms. By 1951, Alexander-Jackson was considered the expert TB microbiologist at Cornell.

In the same year, another American, H.C. Sweany proposed that both the granular and other forms of tuberculosis that passed thru a filter caused Hodgkin’s cancer [6]. This was subsequently supported by studies by Mellon, Beinhauser and Fisher [7,8]. Mellon prophetically warned that tuberculosis could assume both its characteristic red acid-fast forms as well as blue nonacid-fast forms indistinguishable from common germs such as Staphylococci, fungi and the Corynebacteria and that this would surely perplex modern microbiologists.

When organized medicine choose to ignore these studies, Jackson warned that a so-called cure for TB could be as short-lived as it took classical TB rods, for the moment gone underground as a nonacid-fast form, to resurface one day and spring back towards destruction. Although American medicine had no serious time for Alexander-Jackson or her discoveries, it would not disturb her for as long as she focused on tuberculosis and its cousin, leprosy. But when her focus shifted towards Livingston’s cancer germ, it would move to destroy her. She simply posed too great a threat.

By December of 1950 Livingston, who would go on to write over 17 peer reviewed articles by the end of her career, wrote, together with Jackson and four other prominent researchers, what still stands as a milestone on the infectious nature of cancer [9].

At the AMA’s 1953 New York exhibit, participants interest was particularly riveted towards an exhibit of Livingston’s cancer germ, live. The press, muzzled by Sloan Kettering’s head, Cornelius Rhodes, was not allowed to interview or report on this exhibit. Above, the cancer germs seemed indestructible, surviving a five-day experience of intolerable heat from closed-circuit microscopy [1].

As Livingston and Jackson’s work on the cancer germ became more and more convincing, her opponents surfaced and became more and more vocal.

Also with recognition, came visitors. One a pathologist from Scranton, Dr. George Clark, told Livingston he had cultured Dr. Thomas Glover’s famed cancer germ from human cancer and developed metastasizing tumors in animals from it.

Clark assured Livingston that Glover was on to the same bacterial pathogen that she was. For more than two hundred years, the same organism had been discovered and rediscovered, named and renamed, each discoverer adding to what was known about the cancer germ, but thus far to no avail.

Clark knew Glover as part of an investigative team of the US Public Heath Service headed by George W. McCoy in 1929. Glover had just become too well known to be ignored. His cancer serum was working.

Much was at stake. The Country was already committed to the idea that cancer could not possibly be an infectious disease, and Glover was saying that he had already isolated the cancer germ.

Actually, he had not, but few would believe that it was really his young, tobacco-chewing assistant, Thomas Deaken who had isolated it. Deaken worked his way up New York’s health and hospital system from the most menial positions to laboratory assistant. With neither formal medical or scientific training, this laboratory assistant nevertheless learned laboratory protocol [10]. Incredibly Deaken engineered a geranium based culture medium, managing to grow out acid-fast, tubercular bacteria. Then he inoculated mice and dogs, producing cancer with metastatic spreadin every case [10]. Sometime between 1917 and 1918 Thomas Daeken, laboratory assistant, produced a specific anti-cancer sera by injecting horses with the human cancer germ. Moreover, the sera worked whether in prevention or cure of his cancerous laboratory animals. But Glover had come to the point where he needed someone to lend credibility to his work, and that someone, came in the form of Dr. Thomas J. Glover of Toronto.

It will always be to Glover’s credit that he saw the importance and application of Deaken’s work from day one. A contract was quickly drawn up and executed. Glover rushed back to open a Canadian cancer clinic in Toronto. The serum worked in many but not all cases; but as Glover’s reputation grew, so to did the interest in him of Canada’s organized medicine. A subpoena giving him 21 days to submit a full presentation of his treatment was issued. But Glover was not cooperating. Glover was in trouble and would soon be chased out of Canada [10].

By 1926, and now in the US, Glover published Progress in Cancer Research, presenting over 50 cases, most of which went into remission with Glover’s Serum [11]. It sparked additional notoriety, both here and abroad. In 1929, Livingston’s friend Dr. George Clark joined Dr. George McCoy, then head of the Hygienic Lab of the US Public Health Service. Their intended destination: Glover’s laboratory, now at New York’s Murdock Foundation. Glover was under investigation and McCoy wanted him to repeat his work, this time under Health Service surveillance and in Washington. Glover complied, and he and his team went to the nations capital to prove their case at what was to one day become the National Institute of Health.

McCoy, the investigator, impressed by Glover’s work, rather than come down on Glover, instead issued a 1937 letter to Surgeon General Parran, which spoke in glowing terms of the great importance and significance of Glover’s cancer findings.

Soon thereafter, McCoy was abruptly and mysteriously replaced by Dr. R.H. Thompson. Parran, a product of organized medicine, had a definite agenda. The question before him was whether to publish Glover’s now finished Washington report or not and Parran, despite continued committee approval, was not about to, sending Glover into a cold rage which ended with him walking away from Washington to publish independently.Meanwhile, Glover’s serum, which had helped and saved so many was subjected to cursory animal studies and a review without clinical trials before being condemned by Government agencies.

Glover would eventually return to Canada, but he would never again answer questions as to just what had happened in America.

Virginia Livingston now went specifically after breast cancer. Thirty sterile cancerous breasts were transported from operating room to lab. Cancers were isolated from each breast and when axillary tissue from under the arm was supplied, the cancerous portion was cut from this too. Livingston and Jackson found the cancer germ everywhere, and in the case of underarm glands, even when the pathology report was negative, the cancer microorganism surfaced [1].

Champion of toxic chemotherapy, Cornelius Rhoads replaced Ewing at Sloan. Rhoads, head of chemical warfare during the Korean war, was deeply committed to chemotherapy and the huge grants it brought from the pharmaceutical industry.

It is poorly recognized that the chemotherapy or “chemo” used against cancer began as a weapon of mass destruction par excellence [12]. When the Axis folded, nitrogen mustard, declassified, first came under real medical scrutiny for cancer. Initially evaluated for lymphosarcoma in mice, human studies soon followed as more and more variants of nitrogen mustard were concocted and tried [12].

Other related classes of chemotherapeutic agents followed and so did their repercussions. Most had the potential to cause a second entirely different cancer [13]. Even tamoxifen for breast cancer was associated with a two to three-fold increased risk of cancers of the lining of the uterus (endometrial), some of which were high grade with a poor forecast [14].

Nevertheless, Cornelius Rhoads remained committed to the treatment, and at the same time prepared a series of major roadblocks to stop Livingston.

In 1950, he barred her from presenting her paper on the cancer germ at the New York Academy of Sciences by discrediting Irene Diller, the symposiums sponsor, chief-editor of the respected journal Growth, and a prominent cancer researcher. Diller, like many, had accepted a gift from a pharmaceutical house at one point. Livingston came across Diller in a Life Magazine article which talked about a Philadelphia cancer researcher who was observing strange fungus-like filaments protruding from cancer cells. Livingston and Alexander-Jackson convinced her that her fungal forms (the prefix – myco in mycobacteria denotes a germ with fungal properties) were part and parcel of the cancer microbe, and that crucial to its identification was acid-fast staining.

Dr. Eleanor Alexander-Jackson’s elation over the groups infectious breast cancer findings came to an abrupt halt when she was informed by her private physician Frank Adair that she too had it. A radical mastectomy was done at Sloan on Adair’s advice.

While anxiously waiting for the outcome, Dr. Virginia Livingston heard her name paged on Sloan’s overhead. Rhoads wanted to speak to her regarding Jackson’s ongoing surgery. It was urgent. Alexander-Jackson was still in the operating room and the radical mastectomy had been done. In Rhoads office, the two adversaries faced off. incredibly, Rhoads was after permission to go after a cancerous lymph node deep in the middle of Eleanor’s chest. Livingston bristled.

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“We have been looking for a tumor such as she has.” said Rhoads.Apparently a radical was not enough. He was seeking permission to try a new surgical technique which went after the deep chest node. Livingston had had enough. Just the thought of the cruel, disfiguring procedure made her sick.

“Not on your life.” She shot back, as she left [1].

By 1965, Edith Mankiewicz, Director of labs at Montreal’s Royal Edward Chest Hospital and assistant professor of bacteriology at McGill, by examining human cancer tissue, established mycobacteria-like germs inside cancer [15]. In the bibliography of one of her landmark papers is reference to a personal communication with Dr. Eleanor Alexander-Jackson. One of the cancers under Mankiewicz’s trained eye was lung cancer. Lung cancer,or bronchogenic cancer, was first reported in the nineteenth century at a time when it was practically unknown-while mycobacterial disease of the lung, primarily tuberculosis, was so rampant as to be called ‘white plague’ or in certain circles: ‘captain of the men of death.’ By the middle of the seventeenth century, one in five deaths was due to tuberculosis and at the end of the nineteenth century, there was fear that it would destroy the very civilization of Europe. So difficult was it to differentiate tuberculosis from the newly discovered bronchogenic cancer that it was only after cases first mistakenly diagnosed as lung cancer were operated on that the benefits of surgical resection of tuberculosis were recognized [16].

Mankiewicz not only showed the cancer germ in malignant tissue but significantly demonstrated how it probably evolved from tuberculosis and related microorganisms when some of the viral phages that lived in them jumped germs, bringing genetic materials which altered the target germs virulence and made them drug resistant. In fact beneath her microscope lay a pictorial of how the cancer germ emerged from TB-like bacilli to create pre-malignant change in mammalian tissue [15].

By 1970, Sakai Inoue, a PhD from Maebashi, Japan and Marcus Singer, a doctor at Case Western’s Developmental biology, completed the single most convincing study of how bacteria cause cancer altogether, with TB-like mycobacteria. Supported by grants from the American Cancer Society and the National Institute of Health, their study used cold-blooded animals, namely the newt or salamander and thefrog. But similar studies showed its applicability to mice [17] and humans [18,19]. Inoue:

 ”An organism similar to the mycobacterium described here has been isolated and cultured from tumors and blood of tumerous mammals, including man, and when injected into miceand guinea pigs, has been reported to yield a chronic granulomatous disease, neoplasm (cancer), or some intergrade.”                     –Inoue and Singer, 1970

Back in the spring of 1953, Sakai Inoue noticed an adult salamander with a hard mass on its stomach. He removed the mass, which turned out to be malignant. Then he injected tissue from the mass into healthy animals. Again, cancer developed.

In the work that followed, Inoue and Singer, from electron micrographs, knew that bacteria were involved, bacteria which stained acid-fast……..mycobacteria [20]. Inoue inoculated three other types of mycobacteria, into healthy animals. All came down with cancer, something that did not happen when other germs such as staphylococcus or streptococcus were used. Amazingly Inoue and Singer even noted regressions in some of the cancers, especially if very dilute solutions of the germs were used to initiate them. Furthermore, since cancers stemming from ‘carcinogens’ were structurally identical to mycobacterial induced cancers, the investigators results suggested that such ‘carcinogens’ might merely be factors that activate preexisting infection. The phages inside mycobacteria are viruses known to be activated by carcinogens such as UV light and chemicals [21].

Mankiewicz, five years previously, had shown that these phages, once activated, could cause pre-malignant changes in mammalian tissue [15].

Sakai Inoue and Marcus Singer’s study should have once and for all convinced Virginia Livingston’s opponents of the veracity of her results, and that she was not mistaking common contaminants such as staph. or strept. for the cancer germ. . .but it did not.

It was public knowledge in early 1951 that the Black-Stevenson Cancer Foundation intended to award two huge Black grants of 0,000 towards cancer research and that the first would go to Livingston’s group at Newark’s Presbyterian; with an equivalent amount to go to The Memorial Center for Cancer (now Sloan-Kettering), which Rhoads headed. The trustees having already decided this, the actual allocation was left in the hands of Newark lawyer Charles R. Hardin, but fate intervened.

 Livingston:

“Hardin, the lawyer in charge of allocation, soon would lie dying of cancer at Memorial and while still alive was prevailed upon by design of Rhoads to sign a paper giving Rhoads power over how Presbyterian’s grant was to be spent. And that wasn’t going to include further research towards an infectious cause forcancer.”                   -Livingston, 1972

Still Rhoads was not finished. Livingston, already world-recognized, took her cancer microbe and a guest named George Clark to Rome’s Sixth International Congress for Microbiology, a trip paid for by her husband’s firm as a consultant to British industry. In Rome, Livingston met Emy Klieneberger-Nobel at the Lister institute. Klieneberger-Nobel was a pioneer uncovering bacteria without cell walls which led them to assume many forms [32]. She called them ‘L-forms’ in deference to the Institute at which she worked. Her exploration also covered bacteria with cell-wall breeches. In either case, the resulting germs, called ‘cell-wall-deficient’ assumed many forms (pleomorphic). Livingston immediately saw Klieneberger’s work as clearing a large part of the confusion over her many-formed cancer germ.

Livingston’s trip to Rome’s Congress of Microbiology was punctuated by a stop to visit von Brehmer in Frankfort. Von Brehmer’s vaccination techniques, long respected throughout Europe, were now licensed by the German government.

During the war, Wilhelm von Brehmer’s scrimmage with the Nazi medical establishment went right to the top. Severely criticized for saying that cancer was an infectious disease, the struggle eventually found its way to Hitler himself, who, puzzled, yet interested, ordered an inquiry on the matter at the 1936 Nuremberg Party Conference. Subsequently, the committee formed came down hard on von Brehmer’s views. Nevertheless, unperturbed, he somehow persisted into the legendary status he now maintained.

Big names began to join the conference, including Nobel Laureates Fleming and Waksman. By the time Virginia Livingston returned to the States, the Rome conference had been highlighted by several news services. Beginning with the New York Times and The Washington Post, other papers quickly followed suite: the cancer germ had been found. Reaction quickly followed. At The New York Academy of Medicine, spokesman Iago Gladston, fresh from executive session, held his own sort of news conference:

“This is an old story and it has not stood up under investigation. Microorganisms found in malignant tumors have been found to be secondary invaders and not the primary cause of malignancy.”- Livingston, 1972.

Livingston returned to Newark. Her Chief, James Allison, contacted her with the bad news. Since they had lost Black-Stevenson funding, he wanted her to close up Presbyterian’s research and move back to Rutgers’s home campus in distant New Brunswick. And in still another cost-cutting gesture, she was informed that her close friend and associate Eleanor Alexander-Jackson would have to go. Shocked, Livingston made arrangements to leave Rutgers altogether. Barely unpacked from Europe, Livingston’s husband would now be hounded by the IRS regarding where they got the funds for the European trip. Someone had implied the money came from his wife’s grants. This did not bear out and the couple demanded to know who had instigated the inquiry.

“Someone high up in New York in cancer.” The IRS agent replied [1].

 

By 1925 Mayo’s Charles Mayo became interested in Erwin Smith’s discovery of cancer in plants, called crown gall. Livingston and Jackson, sensing a possible link between Smith’s work and their own, went to the Bronx Botanical Garden to request cultures of Bacterium tumefaciens, the plant cancer germ he had discovered. No mere accident led Virginia Livingston towards Smith’s work. Smith stained his plant cancer germ with Fuchsin, long used to spot tuberculosis. And Smith’s bacteria, like Livingston’s, had many shapes. He had stumbled across B. tumefaciens in 1904, when he received some New Jersey daisies with overgrowths superficially resembling olive tuberculosis, a known disease of plants, but which proved to be plant cancer.

Smith had long suspected a bacterial cause for human cancer and criticized pathologists for drawing:

“Too sharp a demarcation between malignant tumors, on the one hand, where the cells of the animal or human host, acting under some unknown stimulus are responsible for the tumerous growth and granulomata (benign tumors) on the other hand, such as tuberculosis and actinomycosis, where a visible microbe isresponsible for the primary tumor, and the direct migration of this microbe for any secondary tumors that may appear.” -Rogers, 1952

Smith’s conclusion:

“At the bottom, I think the distinction between such a disease, for example as tuberculosis or leprosy and malignant tumors is not as sharp as some histologists have been inclined to believe”.   -Rogers, 1952

It could be said that at one time the entire medical and scientific community was set on fire by Erwin Frink Smith’s discovery of the bacteria that caused plant cancer. Twice honorably mentioned in The Journal of the American Medical Association, their Editorial “Is Cancer of Infectious Nature?” mentions how Smith’s work made “a very strong case in favor of his view of the infectious cause of cancer in general.” (JAMA, 1912)

By 1921, Margaret Lewis, of the Livingston Network, approached Frink Smith regarding her planned chicken inoculations with B. tumefaciens. Lewis would go on to elicit the cancer sarcoma from chick embryos using B. tumefaciens.

On January 31, 1925, an English abstract in the authoritative German Kinische Wochenschrift, written by Ferdinand Blumenthal, trapped Smith’s attention. Blumenthal, with assistants Meyer and Auler had shown that human cancer bore a microorganism closely resembling tumefaciens which in turn caused malignant tumors in plants as well as animals, complete with spread or metastasis.

Paula Meyer had worked with Friedlander on the human cancer germ since 1923. Her particular discovery was of a bacteria inside breast cancer which she called PM for Paula Meyers. She had also discovered closely related strains from 15 other human cancers. Smith examined stained slides of Meyer’s cancer germ from human breasts. It looked much like B. tumefaciens. Meyer’s germs were short rods, single or paired, and they stained with the same Fuchsin that he had used [22].

Moreover, when Blumenthal and Meyer inoculated their human cancer germ PM into plants, the tumors looked exactly like crown gall. That PM could produce plant cancer was now for Erwin Frink Smith beyond a shadow of a doubt. But it could not be B. tumefaciens itself, because no strains that he had tested grew at body temperature in warmblooded animals. His conclusion: that human cancer was probably due to some other microbe, possibly a mycobacteria, that had similar chemical activities to B. tumefaciens.

The only time that Dr. Florence Siebert, long part of established medicine, ran into resistance and suppression, was when she decided to have a closer look at Livingston’s cancer germ. One of America’s finest Ph.D. – Biochemist’s, while still at Yale she resolved the mystery of the many fevers coming from distilled water for injection and thought to be caused by fever-producing ‘pyrogens’, quickly proving that these were in fact bacterial contaminants. Having solved the mystery of pyrogens, Siebert was asked by Dr. Esmond Long to stay on at the University of Chicago to develop the Tuberculin skin test. Long suggested a European trip to learn techniques practiced on the continent [23]. At thePasteur Institute of Paris, Seibert exchanged ideas with Boquet, Calmete and Guerin: the three investigators who presented to the world its only recognized vaccine for tuberculosis, called BCG [23]. Seibert returned to the US and when Long left Chicago to head laboratory operations at the Henry Phipps Institute in Philadelphia, she accompanied him.

By 1903, Henry Phipps, wealthy partner of Andrew Carnegie, sought a charitable outlet for his wealth. He then joined Lawrence F. Flick, a doctor with a vision to open a center solely dedicated to the study, treatment and prevention of Tuberculosis.

Still working off grants from the National Tuberculosis Association, Seibert was asked at Phipps to continue her work for a skin test using Koch’s original Old Tuberculin (OT). Seibert refined and purified the protein in her TB skin test. She named it PPD-S, both because it was a purified protein derivative and was intended to serve as a standard (S) for the US Government, which it eventually became. Then, after 30 years in tuberculosis research, Seibert turned towards cancer. In 1948, Margaret Lewis of Philadelphia’s Wistar Institute asked Seibert to do a nucleic acid analysis on Wistar rat tumor extracts, which Seibert agreed.

Next, Irene Diller, who networked extensively with Livingston, asked Seibert to look at her slides of the cancer microbe. Seibert relates what she saw:

“I saw tiny, round, coccoid organisms, many of which were magenta in color. The slides had been stained with Ziehl-Neelsen reagent, which we regularly used to stain our tubercle bacilli red. When I learned that she had isolated them from a rat tumor and could do so regularly from tumors in general, as well as from blood of leukemic patients, I asked,”Could you find them in the rat sarcoma tumorI am studying?”    -Seibert, 1968

Diller agreed to try. Lewis allowed Seibert to forward the tissue sections. The results came back. The same cancer germ appeared. Seibert immediately saw the implications:

“This looked terribly important to me, and I was thenceforth willing to do whatever I could to help in this promising field. We did help by studying the immunological relationship to our tubercle bacilli, as well as to the “atypical” bacteria closely related to our tubercle bacilli.” – Seibert, 1968

Seibert was even more impressed with how Diller, following the footsteps of Livingston and Jackson, proved, thru Koch’s postulates, that her germ was the cancer germ:

“It is based on her (Diller’s) work that I am willing to say I believe she has foundthe cause of cancer, which I think no one can refute, and this work should be welcomed and confirmed by other cancer researchers, and not be ignored, even in view of the great stir at present about viruses.” -Seibert, 1968

Florence Seibert joined Livingston’s crusade in earnest in the 1960s, turning her cancer organism over to Frank Dunbar, chief of laboratories at the Southwest Tuberculosis Hospital in Tampa. Dunbar’s conclusion: her multi-formed germ did not belong to his groups of known “atypical” mycobacteria,even though they did have some of the properties of the mycobacteria [23].

Eventually Virginia Livingston gained university affiliations in San Diego working out of the University of San Diego with Dr. Gerhard Wolter of nearby San Diego State. In 1970, Wolter and Livingston discovered actinomycin-like compounds produced by the cancer germ, one of which, Actinomycin D or Dactinomycin, depite its toxicity, was being used in cancer. Livingston was aghast that her own discovery was being used this way. She cautioned that not only did actinomycins arrest the maturation of cells and inhibit the immune response but that they also inhibited enzymes and decreased hormone levels, stimulating the body to increase its hormone production [1].

She was puzzled as to why anyone would want to use a devastating substance like Actinomycin D for the subsequent treatment of cancer. Yet it was being done. Even more disturbing was the way in which organized medicine was responding to the hormonal disruption in the body caused by her cancer germ.

By 1966, Charles Huggins of the University of Chicago went to Stockholm and received a Nobel Prize for determining the effects of sex hormones on cancer that had spread. Following this, the practice of castrating cancer victims came into vogue. Consequently, someone came to the conclusion that if castration helped initially, any recurrence would better be treated by cutting out the adrenal glands, housed on top of each kidney.

And since this never produced earth-shaking results, a new procedure was devised to cut through the nose and remove the pituitary-the master gland of the body, lodged near the brain. Virginia Livingston had established that abnormal hormonal stimulation was coming from the toxic materials and hormonal derangers manufactured by her germ. In response America was chopping out the glands of its cancer patients.

In The Cancer Microbe, Dr. Alan Cantwell Jr. acknowledged the invaluable help of four women who pioneered the early microbiology of cancer: Virginia Livingston, M.D.; Eleanor Alexander-Jackson, PhD; Florence Siebert PhD and Dr. Irene Diller [24]. Cantwell grew up reading that all germs responsible for the important diseases were supposed to have been already discovered. But much to his dismay, once a physician-researcher, he encountered the one left out: Livingston’s cancer germ. And although he knew that the many-shaped germ had long been considered a mere contaminant or secondary invader or even non-existent, Cantwell, like Seibert, knew better. Cantwell first contacted Virginia Livingston thru the suggestion of a colleague who had heard her on radio and immediately sensed their common ground, which was, by then, the acid-fast bacteria found in Scleroderma and cancer. Despite her meticulous research, Cantwell knew that Livingstone had already been branded by traditional medicine as a charlatan, leaving what he thought to be perhaps the major discovery of the 20th century largely discredited [24].

By 1971, Cantwell had published on Scleroderma in the highly respected Archives of Dermatology and had no further intention of pursuing Livingston’s germ. Livingston, Jackson, Diller and Seibert had each drawn considerable fire from the medical establishment and despite Livingston’s persistent overtures towards him, there was no way he wanted in. By 1974, Lida Mattman’s Cell Wall Deficient Forms [25], reconfirmed for Cantwell as well as others that many bacteria, but especially tuberculosis and the mycobacteria existed naturally in many forms – a cycle of growth which involved “cell-wall-deficient forms” ranging from viral look-a-likes to bacterial forms to granules and then on to larger globoid shapes. But most physicians and laboratory scientists were being taught little about cell-wall deficient bacteria.

Cantwell’s silence threshold was exceeded forever when he again saw the cancer germ in the skin of the chest wall of a young woman who had lost both her breasts to metastatic cancer. Removing this patients skin lumps, Cantwell and colleague Dan Kelso at first cultured Staph. epidermiditis, a common contaminant. But as their cultures aged, the seeming Staph cocci became large globoids, rods and yeast-like forms – with acid-fast TB-like granules everywhere [25].

Tracking down specimens of the woman’s original cancer, removed a year earlier, Cantwell not only isolated the variable acid-fast cancer germ in the tumor itself, but in surrounding specimens taken from the woman and thought by pathologists to be normal. This in effect established that the germ existed in the victims tissues before it became cancerous.

In a series of peer-reviewed, penetrating articles, Cantwell found the cancer microbe in three other cancers: Hodgkin’s, Kaposi’s cancer of the skin and a rarer skin cancer called mycosis fungoides.

It became obvious to Dr. Alan Cantwell after twenty years of microbe hunting that the old tenets of microbiology were not much use when it came to showing an infectious cause of cancer. In man as well as in nature, bacteria were constantly changing forms and evolving in their lifetime. The cancer microbe, unstable by nature, was no exception [25]. But 25 years after removing the metastatic breast nodules from the skin of a young mother and finding them variably acid-fast, there remained no cure for a germ which though tuberculosis-like, seemed indestructible. And a germ without a cure, as shown by the mixed reception to Koch’s discovery of tuberculosis, even decades later, fostered it’s own resentment and disbelief, a resentment and disbelief which Virginia Livingston never stopped facing.

“It seems to me that it is entirely rational to state that the reason the BCG vaccine is effective not only against tuberculosis, but leprosy as well as cancer is because of the fact that the cancer germ is closely related to the BCG since it is in the same family, the Actinomycetales.          -Livingston, 1972

When Florence Seibert met Boquet, Calmete and Guerin in Paris to discuss their BCG, the only recognized vaccine for tuberculosis in the world, made from cow or bovine tuberculosis, none of them had any idea that it would one day be used against cancer. But in fact, currently, this diluted vaccination of Mycobacterium bovis or cow tuberculosis is the most effective treatment for transitional cell carcinoma, a cancer of the urinary bladder. Moreover, BCG is the most successful therapy of its kind, called ‘immunotherapy’ [26]. Within ‘immunotherapy’, it soon became fashionable to suppose that BCG or cow tuberculosis somehow ‘bolstered’ the immune system, but noted immunologist Steven Rosenberg held that the immune system was highly specific. One immune stimulant such as BCG should not stimulate a response from another immune stimulant, cancer [27].

The precise mechanism as seen by a 1993 University of Illinois study was that initially cancer cells seemed to eat (or phagocytize) and kill the Mycobacteria bovis in BCG. But then, suddenly, the cancer cells too died. Although investigators in the study admitted the relationship wasn’t clear, a strong ‘tumoricidal agent’, inside the Mycobacteria was pointed to [28]. Livingston felt that investigators were probably unwittingly looking at was a common phenomena in nature known as ‘lysogeny’. Lysogeny is what happens when one colony of a similar bacteria kills another by hurling viral phage weaponry towards it, without itself being harmed.

By the late 1970s Virginia Livingston could no longer ignore Chisato Maruyama of Japan and sent John Majnarich of Seattle’s BioMed Laboratories to Japan to have a closer look. In 1935, Maruyama, of the Nippon Medical School began to develop a vaccination against tuberculosis which turned out to be good against cancer. The Maruyama vaccine was similar to BCG, but instead of using cow tuberculosis as its base, the Japanese version used human tuberculosis.

Chisato Maruyama had long noted that patients with either the Mycobacteria tuberculosis or leprosy seldom had cancer [33]. By the 1970s Maruyama’s vaccine was proving quite successful in that he claimed that half of the 8000 cancer patients he had treated had benefited [29].

By the early 1970s Virginia Livingston, badly beaten by the medical establishment, was ready to launch a counterattack in the form of a fascinating study which showed that her cancer microbe secreted humanchoriogonadotropic hormone (HCG) – a growth hormone long associated with cancer. Initially, despite laboratory evidence to the contrary, her contention that a bacteria could produce a human hormone was not believed. But then reports from traditional bastions such as Allegheny General, Princeton and Rockefeller University confirmed her findings.

Livingston believed that this growth hormone, secreted by her cancer germ built up uncontrollably to stimulate tumor growth, turning normal cells into malignant ones when either the body’s immune system was weak or essential nutrients were deficient. Dr. Hernan Acevedo of Allegheny, in fact, showed that all cancer cells had the hormone [30].

Livingston’s discovery, a medical milestone, gave further impetus to a microbial theory of cancer with well over a century of research behind it. Yet despite this, the premise behind an infectious cause was stubbornly refused by orthodox medicine.

Virginia Livingston was past 80 when she died on June 30th, 1990. Just months before, a subpoena was issued to her prohibiting her vaccinations, made from the patient’s own cancer germ (autogenous), with which she had had great success. Following this, her vaccine was stigmatized as an “unproven method” in the March–April 1990 issue of CA – The Journal of the American Cancer Society[31] with references to her mistaking several different type of bacteria, rare and common for a unique microbe. This despite droves of research papers establishing mycobacteria as either coming before or coexisting with cancer. Ironically, Acevedo, who could not stop lauded her discovery that the cancer germ could manufacture human growth hormone was instrumental and key to the society’s damaging conclusion.

Yet when questioned by this author approximately a decade later, Acevedo admitted that he had ignored acid-fast forms which were indeed present in the cancer preparations Livingston sent to him. He felt these irrelevant, and mentioned that besides, the technology was not available at the time to pursue these acid-fast forms further.

On such fuzzy logic, it seemed that perhaps the most important scientific cancer lead in this or any other century was buried.

The striking analogy between cancer and tuberculosis was noticed long before the tubercle bacillus was discovered. In 1877, Sir John Simon clearly

pointed out this analogy and in fact argued very strongly in favor of a microbial origin of cancer. But by 1910, certain American medical powers did an 180 degree rotation, deciding that cancer was not caused by a microbe and that anyone who thought otherwise was a heretic, a charlatan or a quack.

But Virginia Livingston was none of these. Rather she was a symbol of painstaking research and dedication at the height of post World War II American medical technology. Opponents of Livingston said that she saw “contaminants” of a group of commonly encountered germs. But Florence Siebert, an expert on contaminants who standardized the present day tuberculin skin test for the US government, saw no contaminants present and Dean Burk, then Head of Cell Chemistry at the NCI went so far to say that Livingston’s cancer germ was as real and certain as anything known about cancer [29]. Yet in the subsequent suppression of Livingston and her many colleagues by the medical establishment a picture emerges, and it is not a very pleasant one.

Virginia Livingston gained international status when she discovered that her cancer germ produced human growth hormone, long associated with malignancy. However, at first even this was not believed. Had she gained the same stature regarding identifying the cancer germ itself, by today there probably would be no cancer. At this time there is admittedly no cure for Livingston’s cancer germ. Suppression led to its own disinterest in cure and each year a multitude must suffer and die as a result.

 

[1] Livingston, Virginia Wuerthele-Caspe, Cancer: a new breakthrough, Los Angeles: Nash Publishing; 1972.

[2] Ewing J. Neoplastic diseases. 2nd ed. Philadelphia: W.B.Saunders; 1919.

[3] Hunter D. The diseases of occupation. 6th ed. Boston: LittleBrown and Company; 1978.

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[7] Beinhauer LG, Mellon RR. Pathogenesis of noncaseatingepitheloid tuberculosis of hypoderm and lymph glands. Arch Dermatol Syph 1938;37:451–60.

[8] Mellon RR, Fisher LW. New studies on the filterability of pure cultures of the tubercle group of microorganisms. JInfect Dis 1932;51:117–28.

[9] Livingston V, Alexander-Jackson EA. Cultural properties andpathogenicity of certain microorganisms observed fromvarious proliferative and neoplastic diseases (published under Virginia Wuerthele-Caspe). Am J Med Sci 1950;220:636–48.

[10] Boesch M. The long search for the truth about cancer. NewYork: GP Putnam’s Sons; 1960.

[11] Glover T, Scott M. A study of the rous chicken sarcoma No.1. Can Lancet Practioner 1926;66(2):49–62.

[12] Goodman LS, Gilman A. The pharmacologic basis of therapeurtics. 5th ed. New York: MacMillan; 1975.

[13] Skirvin JA, Relias V, Koeller J. Long term sequelae of cancerchemotherapy. Highlights Oncol Practice 1996;14(2):26–34.

[14] Pukkala E, Kyyronen P. Tamoxifen and toremifene treatmentof breast cancer and risk of subsequent endometrial cancer: a population-based case-control study. Int J Cancer2002;100(3):337–41.

[15] Mankiewicz E. Bacteriophares that lyse Mycobacteria and Corynebacteria and show cytopathogenic effect on tissuecultures of renal cells of Cercopithecus aethiops. Can Med Assn J 1965;92:31–3.

[16] Dubos R. The white plague: tuberculosis. New Brunswick,NJ: Man & Society Rutgers University Press; 1987.

[17] Aaronson JD. Spontaneous tuberculosis in salt water fish. JInfect Dis 1926;39:315.

[18] Wuerthele-Caspe VE, Alexander-Jackson E, Smith LW. Someaspects of the microbiology of cancer, J Am Woman’s Med Assoc 8:7.

[19] Alexander-Jackson EA. A specific type of microorganismisolated from animal and human cancer. Bacteriol Org Growth 1954;18:37–51.

[20] Inoue S, Singer M. Experiments on a spontaneously originatedvisceral tumor in the Newt, Triturus pyrrhogaster.Annal NY Acad Sci 1970;174:729–64.

[21] Lwoff, A. Biologic order (Karl Taylor compton lectures),Cambridge, MA: The MIT Press; 1962.

[22] Rogers III AD. Erwin Frink Smith: a story of North Americanplant pathology. Philadelphia: American Philosophical Society; 1952.

[23] Seibert FB. Pebbles on the Hill of a Scientist, in: Florence B.Seibert, author/publisher, St. Petersberg, FL 1968.

[24] Cantwell Jr A. The cancer microbe. Aries Rising Press; 1990.

[25] Mattman LH. Cell wall deficient forms – stealth pathogens.2nd ed. Boca Raton, FL: CRC Press; 1993.

[26] Schneider B. Specific binding of Bacillus Calmette–Guerinin urothelial tumor cells. In vitro World J Urol 1994;1216:337–44.

[27] Rosenberg SA, Barry JM. The transformed cell/unlockingthe mysteries of cancer. New York: GP Putnam’s Sons;1992.

[28] Devados PO, Klegerman ME. Phagocytosis of Mycobacteriumbovis BCG organisms by murine S180 sarcoma cells. Cytobios 1993;74(296):49–58.

[29] Martin W. Medical heroes and heretics. Old Greenwich, CT:The Devin Adair Company; 1977. 

[30] Acevedo H. Human choriogonadotropin–like material inbacteria of different species: electron microscopy andimmunocytochemical studies with monoclonal and polyclonal antibodies. J Gen Microbiol 1987;133:783–91.

[31] Congress of the United States Office of Technology Assesment.Unconventional Cancer Treatments US Govt PrintingOffice, Washington, D.C; 1990.

[32] Klieneberger-Nobel E. Origin, development and significanceof L-forms in bacterial cultures. J Gen Microbiol 1949;3:434–42.

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This cancer therapy is based on Natures way of getting rid of cancer. It simulates the life condition of the longevity populations of this world, all of which seem to have many factors in common. These people, many of which live well over 120 years in excellent health, are almost exclusively found in high altitudes of 2000 m (7000 ft) and above. They breathe clean air enriched with tiny amounts of ozone. They drink pure mountain water that flows right of the glaciers. They grow their own food that is rich in vitamins and minerals. Their stress level is low and they are in harmony with their environment.
 
Their spiritual beliefs demand from them to respect all other living beings. It is interesting to note that of the three people with the greatest longevity, two – the Hunzas in Northern Pakistan, and the Abkhazians in the Caucasian Mountains of Georgia near the Black Sea – are devout Muslims, the third, in Vilcabamba, Ecuador, mostly follow Native American Indian animistic beliefs.The first unusual ingredient of the environment of the longevity population – ozone is highly activated oxygen consisting of three atoms. This triatomic oxygen is the most powerful purifier of the Earth and of all living beings. In the simplest terms, ozone is capable of burning all poisonous substances at temperatures between 10 to 40 degrees Celsius (50 to 104 degrees Fahrenheit), as well as killing all bacteria, viruses, and other microorganisms that may contribute to cancers.
 
Ozone is produced by the action of ultraviolet sun light on the oxygen in the air. The higher up we go, the more ultraviolet, and thus, the more ozone. Since time immemorial, it was known that women, who grew up in lowlands, would not get pregnant for at least six months if they moved to altitudes of 3600 m (12000 ft.) or higher. We believe now that ozone naturally prevents a pregnancy until these women are fully acclimatized to high altitudes. In the same way, as ozone temporarily stops the growth of the embryo, it also stops the growth of any fast growing cancer.
We know from the research of Prof. Dr. Otto von Warburg in the 1920s that the cancer cell acts like a plant cell and is dependent for its energy metabolism on lactic fermentation. Fermentation is 19 times less effective than oxidation, the normal energy metabolism of the entire animal kingdom. Fermentation is very sensitive to minute amounts of ozone and there are virtually no cancers observed in people living in altitudes above 2100 m (7000 ft.).
 
All longevity populations live on a diet rich in certain vitamins and minerals that have been proven effective in preventing cancer. Most important among these nutrients are vitamin C (ascorbic acid and ascorbates), vitamin A (retinoic acid and derivatives) and beta-carotene, vitamin E (mixed tocopherols), vitamin D2 from UV irradiation of ergosterol, the high-pH minerals cesium (Cs), rubidium (Rb), and potassium (K), and the trace minerals zinc (Zn), selenium (Se), molybdenum (Mo), and vanadium (V). These nutrients are found in the home-grown vegetables and fruits that are mostly eaten within a few hours after they are harvested. Needless to say, they are grown organically, without the use of harsh chemical fertilizers and pesticides. Most of the drinking water is glacier run-off, called milk of the mountains that is rich in rubidium and cesium. Magnesium (Mg), with calcium (Ca), essential for the transport of oxygen into cells, and potassium (K) with Mg, the most important intracellular electrolytes, are abundant both in green vegetables and drinking water consumed by longevity populations. It is interesting to note that most longevity populations go through prolonged periods of fasts on a yearly basis, be it during the month of Ramadan or during the leaner part of the year before the crops are harvested.
 
If ozone in higher doses is applied intravenously, it is effective not only to prevent cancer, but to reverse many cancers, especially cancers of the lungs, liver, pancreas, and metastatic cancers to the bone, as is well documented in the medical literature. Doctor A.K. Brewerâs high-pH therapy, using high doses of cesium (or rubidium), and enhanced by weak acids such as ascorbic acid (vitamin C) and retinoic acid (derived from vitamin A) , as well as ampholytic elements such as zinc and selenium, has been proven effective in virtually all fast growing cancers, both after oral and intravenous application. This is further enhanced by amilonitriles contained in apricot pits that are part of the regular diet of the Hunzas, and may also be applied intravenously in the form of Laetrile.
 
The intravenous form of the enhanced high-pH therapy also contains generous amounts of the intracellular electrolytes magnesium and potassium. The dosage of the I.V. therapy is adjusted to reduce virtually all smaller cancer accumulations (up to 20 or 30 cm diameter), providing that they are fast growing tumors, by one to two centimeters per day (2/5 to 4/5 per day). Large tumor masses are reduced with the I.V. therapy by 500 to 900 grams per day (1 to 2 lbs. /day) to prevent an over-loading of the bodies metabolism and excretion with tumor breakdown products. The critical factor is the kidney and liver function of the cancer patient before the enhanced high-pH therapy is started. One important thing to keep in kind is that, though the enhanced high-pH therapy was seemingly effective, some patients may still succumb from the adverse effects of cancer chemotherapy, or from complications of radiation or surgery undergone previously. Also, if a cancer patient, after the tumor disappeared with the high pH therapy, does not change his lifestyle and eating habits, cancers may develop again in his or her body.
 
How does all of this work?
Most orthodox cancer chemotherapy proffers only a large number of unproven theories and in almost all cases shortens the survival after severe suffering form its adverse effects1. On the other hand, the enhanced high-pH cancer therapy is proven effective by clinical and experimental studies that filled over two thirds of Supplement 1, to the major peer-reviewed medical journal Pharmacology, Biochemistry, and Behavior, of December 1984 [21 Suppl 1: 1-135]2.
 
Also, on this therapy, almost all patients, no matter how far gone or suffering from the adverse effects of chemotherapy and/or radiation, will feel much better within a few days. Particularly, cancer pain, even if unresponsive to the most powerful pain killers, in most cases disappears within only a few hours after starting the cesium.
 
Any symptoms connected with this therapy, particularly from the I.V. ozone, are almost always the result of a healing crisis, well known to homeopaths for over 200 years. These symptoms may be quite uncomfortable but subside in most cases within a few hours, and many patients report that afterwards they felt better then ever before in their lives.
 
1 See Appendix II to the author’s two Cancer Articles: “Nutrients & Cancer” and “Cesium Therapy in Cancer Patients”, Pharmacol Biochem Behav 1984; Suppl 1: 7-10 & 11-3, respectively.
 
2 See Appendix I to and also the author’s two Cancer Articles of 1984.
 
In the following we will briefly explain how cancers form (i.e., carcinogenesis) and how the enhanced high-pH cancer therapy transforms cancer cells either to normal cells or makes them disappear altogether.
 
Professor Dr. Otto von Warburg, double Nobel laureate, in medicine and biochemistry, in the 1920s discovered the fundamental mechanism of carcinogenesis. When certain cancer-causing chemicals (carcinogens) attach to the cell membrane, the oxygen carriers calcium and magnesium are unable to enter these cells. The resulting oxygen starvation causes these cells to regress to anaerobic (i.e., without oxygen) metabolism [which is 19 times less effective than aerobic (with oxygen) metabolism, as was stated previously].
 
The end product of anaerobic breakdown of glucose is lactic acid which renders the cell acidic. This acidosis, in turn, causes the genetic changes that result in the uncontrolled growth of cancer cells. The pH in cancer cells, because of the lactic acid buildup, lowers from about 7.2 to 7.0 (in fast growing tissues) to between 6.8 and 6.0, and in some fast growing metastases to even 5.5. This renders cancer cells extremely vulnerable to ozone and alkalinity which, if applied in minute amounts, either normalizes or destroys them.
 
Specifically, intravenous ozone has the four major effects in cancer patients.
 
One, it removes homeopathically whatever disposed a specific organism to cancer and this causes the healing crisis. This healing crisis may be quite uncomfortable subjectively (though lasting at most a few hours), but there is less than a one in a million chance of serious complications.
Two, ozone removes all toxic and carcinogenic chemicals, amoebas, viruses, bacteria, and other agents from the body that may in some way contribute to cancer and this may be the reason why it seems to be cancer preventative.
Three, ozone inhibits any fast and uncontrolled growth typical for cancer cells.
And four, ozone has a well documented immuno-stimulating effect that helps both with the protection from cancer and with the removal of cancer cells destroyed by the high-pH therapy, enhances the body’s resistance to infections, and boosts longevity.
The more acidic the cancer cells, i.e., the lower their pH, the more vulnerable they are to alkaline, or high-pH, agents. While normal cells are not permeable for cesium or rubidium, and require a transport mechanism for potassium, these three alkalizing elements freely diffuse into cancer cells. This causes the pH to raise in cancer cell; and the higher the pH in the cancer cells, the faster the cancer breaks down. If the intracellular pH is raised to above 8.5, you can actually see the skin wrinkle while you watch over areas where there previously was a superficial cancer tumor, e.g., a breast cancer.
 
This diffusion of alkalizing elements is enhanced by ascorbic acid (vitamin C) and retinoic acid (from vitamin A). Zinc and selenium further enhance the penetration of cesium, etc., by broadening the electron donor capacity of the cell membrane. Zinc and selenium are also powerful immunostimulants, and help with the removal of tumor cells by phagocytotic (lit. cell-eating) neutrophil leukocytes (white blood cells) and monocytes (also called macrophages or â big cell-eaters). Selenium, vitamin E, and beta-carotene are powerful antioxidantts that scavenge dangerous free radicals. Vitamin E also prevents the toxicity of high doses of vitamin A. Molybdenum enhances cancer-destroying oxidases, and vanadium assists with lipid and fatty aid metabolism for faster breakdown of cancers.
 
What is the reality of the 2004 State of the Cancer Treatment in the U.S.A.?
After 35 years of war-on-cancer, and almost $ 90 billion of research funding by the U.S. Government, the cancer death rate in the U.S. of A. increased almost six-fold from 145,000 in 1970, to an estimated 850,000 for 2004. Each insured cancer patient is presently worth between $ 150,000 to $ 500,000 (average about $ 200,000) to the medical profession, hospitals, and the pharmaceutical industry. The out of pocket expenses for insured patients range from $ 30,000 to 100,000, average about $ 40,000, whereas the ULS Cancer Therapy is offered at $ 16,000.00 / €14,000.00. The total national expenditure for cancer management to the premature death of over 800,000 people per year exceeds $ 100,000,000 ($ 100 billion), and, in addition, there are economic losses of the families of the prematurely deceased of perhaps $ 120 billion if their lives had been saved by effective alternative therapies.
 
This total financial investment for patients undergoing the enhanced high-pH cancer therapy is about one-half to one-tenth of the out of pocket expenses of the average insured cancer victim undergoing conventional orthodox cancer therapies. Best of all, the success rate with the enhanced high-pH therapy is consistently much higher and in many cases over 95%, particularly if you are not suffering from severe toxicity of chemotherapy or from radiation damages. And this includes proven incurable (i.e., by orthodox therapies) cancers of the lungs, liver, pancreas, brain, prostate, breast, bones, melanomas, lymphomas, sarcomas, and leukemias.
 
Because of the potential (especially, financial) impact of the enhanced high-pH therapy on the medical/hospital/pharmaceutical industry complex and their most powerful lobby in Washington, D.C., and in many State Governments, this effective, economical, non-toxic treatment can only be offered offshore, i.e., at a location outside the United States. However, every effort is made to have these offshore hospitals properly accredited and to have the costs of the treatments reimbursed by most insurance carriers. The first of these locations is now available in Northern Thailand at a first class hospital for Alternative Medicines that, Insha’Allah, will be upgraded to the point that it is eligible for Blue Cross insurance payments. (Added update) and also at XYZ Wellbeing ReTreat Facility and Research Cancer Centre in  Located in the the beautiful  Cartagena South America. Visit www.xyz-wellbeing.com and go to the why choose us link for more cesium information and cancer research.
 
Therefore, if you, or any of your loved-ones or friends have cancer, even if it was so far ân incurable with chemo, radiation, and/or surgery, please contact The above to see if you may be eligible for the enhanced high-pH therapy. We are committed to one thing only ând to return you to your mental, emotional, and spiritual wellbeing. As long as you faithfully follow the path that we map out for you, you have an excellent chance of emulating the joyous, vigorous longevity of the people who served as the models for the enhanced high-pH therapy. However, it cannot be stressed enough, that the shrinking of a tumor is by far the lesser part of overcoming cancer.
 
Much more important for lasting success is to overcome the cancer personality, defined in the 1960s by Lawrence LeShan, and to embark on an overall healthy lifestyle that equals and excels (by more advanced knowledge) the one the longevity populations. And, perhaps, most important is your will to live and your absolute need to have to accomplish things that must not be left undone by your premature death from cancer. By taking charge of your life in this manner and by taking responsibility by following our leads in all aspects of your life, you will make it possible not only to become free of cancer but remain free of it permanently.

We can only lead you to the Path.
 
It is up to you to walk it and to make sure that everyone around you walks it with you and all the way!

 

No matter what, always keep in kind that, fundamentally, the Lord wills the ultimate outcome of everything in your life. Just as the Lord lead you to the enhanced high-pH therapy to get rid of your cancer tumor, and to the comprehensive Ultralifescience Program for physical, mental, emotional, and spiritual wellbeing, the extent to which you will succeed with it is entirely as the Lord wills. Our promise to you is simply that we will leave no stone unturned to provide for you all the tools for your success in this endeavor.

To your abiding health, vigor, and happiness!

 

__________________________________

Abdul-Haqq H.E. Sartori, M.D.

 

Even if your doctor sends you home to die perhaps telling you “We have done everything we know, there is nothing else we have to offer to help you, except letting you die in peace”.

Did you ever wonder that before about 1900, cancer was a rare disease and that in some parts of the world there is NO CANCER at all? Research that goes back to Dr Otto von Warburg in the 1920s revealed the true nature of cancer and Dr A. Keith Brewer since the 1950, in part through investigation of cancer-free populations, formulated an effective treatment for cancer. This treatment was applied to many cancer patients and further enhanced by Dr Sartori since1980.

Almost all cancers in over 700 patients treated so far with this enhanced high pH therapy, responded within a few days and with I.V. application, daily shrinking of tumors between 1.0 and 2.0 cm can be expected. The only discomfort from this treatment comes from a “healing crisis” reaction that leaves you, after some initial discomfort, feeling better after a few hours or, at most, a day or two. How does this all work? Dr von Warburg found that cancer cells, like plant cells, function without oxygen and thus are very sensitive to oxygen and very strong alkaline elements. Because of the lack of oxygen, cancer cells break down their fuel, glucose, to lactic acid. This causes cancer cells to become acidic (i.e., the pH in the cancer cell is lowered to 6.8, even 5.8) which, in turn, causes them to grow out of control. Alkaline elements, particularly cesium, but also rubidium and potassium can freely enter cancer cells (but not normal cells) causing them to become alkaline or raise the pH in the cancer cell. This raised pH slows down the cancer growth and at a pH of 8.0 all cancer cell growth stops and the cancer cells either die or are turned into normal cells. While we all depend on oxygen to survive, cancer cells die if exposed to oxygen and, particularly, its most powerful form, ozone. People who live very long are free of cancer, is a fact that prompted Dr Brewer to investigate their nutrition and found that their diet contains the alkaline elements cesium (Cs), rubidium (Rb), and potassium (K), and other nutrients that were found to reduce the cancer incidence such as zinc (Zn), selenium (Se), molybdenum (Mo), vanadium (V), and the vitamins A, C and E, as well as amygdalins from apricot pits. After extensive studies of cancer cell cultures, Dr Brewer found the following: Zinc and selenium attach to the cancer cell membrane and make it easier for the cesium and rubidium to enter the cancer cells. Vitamins A and C are weak acids that attract these elements to the inside of cancer cells. Magnesium (Mg) and calcium (Ca) that normally transport the oxygen into cells are depleted in cancer cells. These and other findings were the basis for Dr Brewer to formulate the high pH therapy for cancer. His method was enhanced in the 1980s by adding I.V. ozone (which is the most active form of oxygen), herbal combinations, and other modalities, which made it even more effective.

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Up to 98% of animals with cancers were cured by Dr Brewer’s high pH therapy.

Tests on mice fed cesium and rubidium showed marked shrinkage in the tumor masses of abdominal implants of mammary tumors (“breast cancers”) within 2 weeks. In addition, the mice showed none of the side effects of cancer. Cesium chloride, zinc gluconate and vitamin A were used together to alter growth of colon cancer implants in mice and the use of these compounds was responsible for the disappearance of tumors in 98% of the animals. Sarcoma I implants in mice and Novikoff hepatoma in rats disappeared if the proper ratio between cesium and potassium was maintained. With Dr Brewer’s complete protocol, using cesium (&/or rubidium), potassium & magnesium, vitamins A, C, & E, zinc, selenium, & amygdaline, there was a prompt reduction of all tumors treated by Dr Sartori including lymphomas in cats and dogs, skin cancers in dogs, cancers of the mammary glands, mouth , and esophagus in horses, and cancers of unknown primary in chickens.

Like with all “nutritional” treatments, the principle of the weakest link of the chain holds true, and if even one essential nutrient is lacking, the treatment may fail. In virtually all of over 700 patients with different types of cancer, the enhanced high pH therapy was effective in reducing the tumor mass. Over 90% of these patients were terminal with extensive metastasis and had received maximum conventional cancer treatments. Malignancies treated with this protocol included cancers of the lungs, liver (& gallbladder), pancreas, breast, prostate, colon & rectum, stomach, brain, cervix & uterus, ovaries, testicles, adrenals, kidneys & bladder, of unknown primary, rectovaginal, etc., as well as lymphomas & leukemias, melanomas, & sarcomas & bone. The results with the LSU/ULS Cancer treatment in 100 cancers are detailed in the attached articles. Summary of and Comments on the LSU (now ULS) Cancer Treatment Results. There are several factors that should be pointed out with regard to the data summarized in Table I

(a) Out of over 500 cancer patients treated from 1980 to 1987, only 97 fulfilled the criteria of having been followed up for at least 5 years or until their death. This might negatively bias the number of patients that have died by a factor of up to five since almost all of the over 500 patients were followed for at least 3 months.

(b) According to Arlin J. Brown (AJB), cancer survival statistics as published by the National Cancer Institute (NCI) are not point-to-point, but are determined from the number that can be located 5 years after being diagnosed with cancer (and not even the beginning their first treatment, e.g., at) at NIH/NCI. In cancers with high mortality such as small cell lung cancers (1.0% 5-year survival according to NCI) and pancreas cancers (3.0% 5-year survival according to NCI), AJB found point-to-point survival rates of less than 0.01% and less than 0.05% respectively (perhaps because >99% of the patients had died so long ago that they could not be located anymore).

(c) By far, the majority of the patients seem at LSU were using our therapy as their last resort after all other treatments (both conventional & alternative) had been unsuccessful and most patients were simply sent home to die.

(d) In view of the extremely unfavorable patient population as outlined under (a) through (c), we believe that the results of the LSU treatment are quite remarkable and by far the best offered anywhere in the world.

(e) For reasons beyond the control of the authors, only about 200 cancer patients were treated from 1988 through 2003. In all of these patients, ozone and the minerals and vitamins were applied intravenously (I.V.). The I.V. application of minerals and vitamins proved to be a dramatic improvement in that (i) in virtually all cases, the size/diameter of all fastgrowing tumors was reduced by 1.0 to 2.0 cm (0.4 to 0.8 inches) per day, i.e., a disappearance of a 5.0 cm (2 inch) tumor within four days and of a 10 cm (4 inch) tumor within eight days, and (ii) virtually none of the patients showed any of the side effects frequently encountered with oral vitamin/mineral application such as nausea, diarrhea, abdominal discomfort, possible aggravation of ulcer symptoms, and sometimes even vomiting.

After several cancer patients were successfully treated at the Integrated Medical Center in Northern Virginia from April to July 1998, from mid 1998 until mid2003, government agencies and law enforcement in the U.S.A. virtually completely suppressed the use of the enhanced high–pH cancer therapy by LSU/ULS, and this treatment can now only be offered offshore and far removed from these agencies.

(broken down into the most frequent types/locations of cancers treated) 1. Lung Cancers Of the 18 lung cancers described in this study (of a total of >100), 14 were connected to active smoking, two to passive smoking, one to radon exposure in the home, and one to cadmium exposure at the workplace. Asbestos may have been a factor in one of the active smokers, radon in the home in one of the passive smokers.

Beta-carotene, vitamin A, selenium, and vitamin E from green and yellow vegetables are now recognized as clearly preventative of lung cancer. These vegetables were conspicuously absent from the diet of most of our lung cancer patients. Instead, most of them were eating a meat and potato diet before they started the LSU cancer treatment program. Histologically, 4 patients had epidermoid cancers, 3 had adenocarcinomas, 8 had small cell carcinomas, 2 had large cell carcinomas, and in 2 patients the histologic type was unknown; two of the small cell carcinoma patients also had a lymphoma. All patients had received the full course of orthodox treatment: 6 had surgical resections (3 of the epidermoid-, and one each of the adeno-, small cell-, and large cell carcinomas). All patients had received chemotherapy, and the 6 surgical patients also had received radiation. At the beginning of the treatment, four of the patients were dying on a stretcher, four could walk only with assistance, six were given a prognosis of less than 6 months of survival, and in 4, the prognosis was unknown. The 2 patients with unknown histology who came in dying on a stretcher nevertheless survived 13 and 20 days respectively. The third of the dying patients, with an epidermoid cancer, survived almost 3 months until he died from internal bleeding from an extremely low platelet count. The fourth of the dying patients survived over 5 years and was well in July 1992; he had a small cell carcinoma that generally has less than 1% chance of 5 year survival (less than 0.01% according to Arlin J. Brown). One of the two small cell carcinoma patients who also had a lymphoma is alive and well without any sign of cancer over 10 years after he was barely able to walk into the office with assistance. He is now in excellent health and successfully runs a medical equipment company. The other unfortunately died in a hit-and-run car accident 10 months beyond his given life expectancy and without any sign of cancer at autopsy. One of the adenocarcinoma patients who came in, walking with assistance, responded well for about 2 weeks, then continuously deteriorated, and died after 4 months. The fourth walk-assist patient, with a large cell cancer was treated 4 times and died after 1 year and 8 months. Of the 6 patients who were given fewer than 6 months to live, one epidermoid cancer patient died from cardiac failure after 3 years and 4 months, one of the small cell cancer patients with terminal emphysema died from a combination of pulmonary failure and bronchopneumonia; one patient with adenocarcinoma received 6 treatment series and died from his cancer after 3 years and 8 months; one small cell cancer patient died after 2 years 5 months, one after 4 years 1 month, one epidermoid cancer patient died after 3 years 3 months. One of the factors in the deaths of these patients may have been that at the time of their treatments, the LSU mental reconditioning program (MRP) was far less developed. By using the full, presently available LSU MRP, perhaps at least two, maybe even four of these patients could have been helped. Of the lung cancer patients who survived over five years, four had a small cell cancer, one had a large cell, and one had an epidermoid cancer. 2. Lymphomas Of the 13 lymphomas described in this study (of a total of >60), 9 were lymphocytic (3 males had AIDS, one male had severe rheumatoid arthritis, and one was a Klinefelter syndrome; 4 were females), one female had Hodgkin lymphoma, one male had a T-cell lymphoma, and in 2 males, the histology was not determined. Three patients were dying, 4 needed ambulatory assistance partially because of their enormous tumors, and 3 were given less than a year to live. One of the dying patients with lymphoma of unknown histology died after 17 days from cardiac toxicity of chemotherapy. Another of them, an AIDS patient, died after 7 weeks from aplastic anemia from combined chemotherapies for infections and the lymphoma, given to the patient prior to his coming to LSU. No signs of lymphoma were detected at time of death. One 37 year old dying woman has survived over 10 years without any sign of recurrence after only one series of the LSU treatment.

Of the 4 patients who needed assistance with walking, one AIDS patient is alive and well for over 8 years, has turned HIV negative at the end of one treatment series and his T4 cell count rose from 124 with a T4/T8 ratio of 0.36 to between 1,100 and 1,300 with a T4/T8 ratio between 1.5 and 1.8 for the last 4 years. Within one month, his nodal lymphomas disappeared and none of his previous CNS involvement was detected anymore on a CAT scan. One patient had a huge hemispheric protrusion of his abdomen, very similar to a patient described in Pharmacol. Biochem. Behav., Vol. 21, Suppl. 1, pp. 11-13, 1984. His total tumor mass was estimated to be about 37 kg with about 40 liters of ascites. Within 3 weeks both tumor and ascites were reduced to approximately one half, within 2 months there was only a slight enlargement of the spleen of about 5 cm. The patient survived for over five years without any sign of tumor recurrence. The two patients who had both lymphoma and lung cancer were already discussed under 1.; one of them is alive and well, the other died 10 months after treatment in a hitand- run accident. He had shown no signs of cancer at autopsy. One of the 3 patients who were given less than a year to live, unexpectedly died from a heart attack 10 months after initial treatment. Another died after 3 years and 7 months and did not respond to treatments, except for the initial series. The third patient survived for over 5 years without sign of tumor recurrence. The woman with Hodgkin lymphoma died from aplastic anemia, a complication of her previously received chemotherapy, 1 year and 2 months after treatment onset. The patient with the T-cell lymphoma had come all the way from Osaka, Japan and seemed to respond well to the first treatment series. He returned 5 months later, showed barely any response to the treatment, and died 11 months after the initial visit. Language problems may have been a contributing factor to his death, since we were not sure, whether he and his family had completely understood our instructions. 3. Liver Cancers Primary hepatocellular carcinoma (HCC) or malignant hepatoma is one of the most common malignancies in the world and it is estimated to be responsible for up to 1,300,000 deaths every year. In portions of Africa and Asia, HCC is the most common malignant tumor. It occurs infrequently in the U.S., North and South America, and Europe where it accounts for about 2% of the malignancies. The incidence of HCC is especially high in China, Taiwan, Mozambique, and Singapore. Risk factors of HCC include chronic toxic hepatic injury (20 to 60% in N&S America), cirrhosis (60 to 90% worldwide), chronic hepatitis B infection (20 to 90% worldwide), aflatoxin (especially in Africa and Asia, e.g. from peanut oil), alcoholism, chronic hepatic outflow obstruction (CHOO; 20% in South Africa, 60+% in Japan), male gender (5:1 in high incidence areas, 2:1 in low incidence areas), Asian or Black ancestry (or rather dietary habits). Of the 12 patients listed as having liver cancer (of a total of >50), 8 had primary HCC, 3 had extensive liver metastasis from an occult primary malignancy (OPM), and one patient had intrahepatic biliary cancer (IHBC).The 8 patients with HCC had elevated alpha fetoprotein (AFP) and reduction of AFP below 100 mg/mL was interpreted as an indication of tumor disappearance. Using a cutoff for serum levels of 10 ng/mL, AFP is sensitive for HCC in 70 to 90%. Patients with cirrhosis and chronic hepatitis tend to have elevated AFP levels of usually under 200 ng/mL. Levels of 400 to 1,000 ng/mL are diagnostic for HCC. AFP is also elevated in yolk sac tumors and in a high proportion of other germ cell tumors.

The patient with IHBC and the 3 patients with liver metastasis from OPM had elevated carcinoembryonic antigen (CEA) in the range of 55 to 185 ng/mL at their admission to the LSU cancer treatment program. No colorectal cancer or other primary malignancy was ever found. Elevated CEA levels are found in patients with gastrointestinal, pancreatic, breast, lung, thyroid medullary, and genitourinary carcinomas, as well as in benign disorders including inflammatory bowel disease, cirrhosis of the liver, pancreatitis, and pneumonia. Normal values for CEA are up to 2.5 ng/mL, in smokers up to 5.0 ng/mL. Benign disorders seldom elevate the CEA level above 10 ng/mL. Reduction of CEA levels below 5 ng/mL was interpreted as an indication of tumor disappearance. Of the 12 liver cancer patients, 3 were dying, 3 needed assistance when walking, and 4 were given life expectancies of less than 6 months. 9 had undergone surgery, including the 3 OPM and the IHBC patients; 5 had suffered radiation treatment, and all 12 had been exposed no massive chemotherapy. One female HCC patient, a 32-year-old fitness instructor, had been first seen in the office of a world famous diet doctor in New York City, where she almost died on the table from an imbalanced vitamin-mineral IV. Through almost a miracle she made it to Washington, D.C., lying on a stretcher in the station wagon driven by her husband. Within 2 weeks her massively enlarged liver that had extended over 14 cm below the normal in a scalloped curve that filled about two-thirds of her abdomen, had returned to normal. Her AFP test came down from 2,420 ng/L to 120 ng/mL within 24 weeks. She was well until about 4 years later when she died in a car crash. Unfortunately, the diet doctor never referred any other cancer patient to the LSU clinics. Four more of the HCC patients, and one of the OPM patients, responded very well and survived over 8 years after their initial treatment without signs of recurrence, with AFP and CEA below the cutoff points of 100 ng/mL and 5.0 ng/mL respectively. One HCC patient died from the side effects of chemotherapy within 2 weeks, another within 2 months; one OPM patient shared the same fate after fewer than 3 months. The IHCP survived 2 years and 4 months, after responding moderately well to 3 courses of the LSU cancer treatment. 4. Pancreas Cancer The tumor-associated carbohydrate antigen, CA 19-9, detects about 80% of all pancreatic cancers correctly, compared with 8% of patients with pancreatitis and 1% false positive normal patients. The pancreatic adenocarcinoma glycoprotein, DU-PAN-2,. detects up to 55% of all pancreatic cancers, though in may also be elevated in patients with biliary cirrhosis, gastric cancer, and biliary cancer. In all of our 11 pancreatic cancer patients(of a total of >50), either CA 19-9, DU-PAN-2, or both markers were elevated to a range of 850 to 950 U/mL for CA 19-9, and 300 to 1,200 U/mL for DU-PAN-2 at admission, and reductions of serum levels below 70 or 120 U/mL, respectively, were considered as evidence of disappearance of the tumor. CA 19-9 antigen (detectable by a murine IgG1 monoclonal antibody against a human colon carcinoma cell line) is elevated in 55 to 90% of stomach cancers, 80% of pancreatic cancers, and about 95% of colorectal cancers; in advanced pancreatic cancers it is elevated in 80-90%. In benign disorders including acute pancreatic, hepatobiliary disease, and inflammatory bowel disease, CA 19-9 usually does not exceed 100 U/mL. Normal values of CA 19-9 are up to 36 U/mL. DU-PAN-2 is a mucin-type glycoprotein antigen selected for reactivity against human pancreatic carcinoma cells (detectable by murine monoclomal antibodies). Increased levels occur in many diseases of the liver and hepatobiliary tree including primary biliary cirrhosis, sclerosing cholangitis, hepatitis, cirrhosis, and benign hepatomas, and usually do not exceed 200 U/mL. DU-PAN-2 may also be elevated in biliary and gastric cancer, and in primary hepatocellular carcinoma (HCC). Normal DU-PAN-2 values are up to 60 U/mL. Histologically 10 of the 11 patients had an adenocarcinoma of the pancreas, one had an intrapancreatic bile duct carcinoma (IPDC) that was diagnosed intraoperatively. One patient had both stomach and pancreatic cancer. Eight of the patients had undergone resections and/or exploratory surgery, 10 had suffered from radiation, and all 11 had been given massive doses of chemotherapy.

At the onset of the LSU treatment,
one patient was dying, 3 needed walking assistance, and 6 were given fewer than 6 months to live.

Two patients died from the side effects of chemotherapy within less than 3 weeks including the patient with IPDC. One other succumbed from chemotherapy side effects after 10 weeks. One patient died after about 10 months from an internal bleeding probably not related to cancer. The patient with stomach and pancreatic cancer did not respond well to 3 treatment courses. Nevertheless, they prolonged his life from an expected less than one month to 1 year and 7 months. One patient died after 3 years and 2 months, another after 3 years and 11 months. Nevertheless, the treatment had extended their life expectancy of less than 6 months. Four of the 11 patients survived more than 5 years which compares favorably with a reported 5-year survival rate of pancreas cancer patients of 3% (or less than 0.01% according to Arlin J. Brown). 5. Breast Cancer Six of the nine breast cancer patients (of a total of >40), who are discussed in this report were terminal with widespread metastatic disease, one of them dying, two of them needing walking assistance, and another three with a life expectancy of less than 6 months. In all cases, any detectable primary tumors or metastatic skin tumors either disappeared within 2 weeks or turned from hard, knobby, scalloped, infiltrative cancerous growths into much smaller well-defined, round, and much softer benign cysts with a smooth surface. Unfortunately, two months after treatment onset, one patient died of cardiac failure from doxorubicin toxicity, and one patient died from acute pericarditis-myocarditis from cyclophosphamide less than 3 weeks after treatment was started. One patient responded well to the first treatment course, but had a recurrence after 3 months, and died from pneumonitis. It is possible that an ill-advised treatment course with bleomycin may have contributed to her demise. One patient, a former heavy smoker aged 57 when her treatment began, died after 2 years and 11 months from a myocardial infarction. 5-fluorouracil treatment may also have contributed to her premature death. Another patient who responded poorly to the treatment nevertheless survived 2 years and 2 months, more than 2 years longer than she expected before she started the LSU treatment. The remaining 4 patients survived over 5 years without any sign of recurrence. 6. Prostate Cancers Six of the 8 prostate cancer patients in this study (of a total of >40), had extensive metastatic disease, one of them was dying, two needed assistance with walking, and 4 were given less than 6 months to live. All patients showed elevated levels of prostatic specific antigen (PSA) that ranged from 35 to 235 ng/mL at admission (Normal PSA < 4.0 ng/mL). In benign prostatic hypertrophy (BPH), PSA levels <25 ng/mL are seen. PSA is false negative in about 15% of the prostate cancers. The cutoff point for the disappearance of the cancer was set at 10 ng/mL. Very similar to the results in breast cancer patients, all palpable infiltrating tumor masses in all patients either disappeared or turned into benign, well-defined, cystic tumors of much smaller size. The dying patient succumbed to the side-effects of his chemotherapy 20 days after the beginning of his treatment. One of the severely debilitated patients died after 9 weeks also as a consequence of his chemotherapy. Two patients only partially responded to the treatment. One of these died in a horseback riding accident, the other died after 4 treatment courses 2 years and 5 months after he started the LSU cancer treatment. He had survived almost 2 years longer than was originally expected.

Four patients survived at least 5 years, two of them needed only one treatment course, one of them needed two, and the fourth needed four treatment courses. Their PSA levels were maintained below 10 ng/mL after their treatments were completed. 7. Colorectal Cancers Of the 6 patients in this study with colorectal cancers (of a total of >50), all had elevated values of carcinoembryonic antigen (CEA) in the range of 80 to 280 ng/mL, indicative of widespread metastatic disease; all of them had undergone surgical resections, 4 with colostomy, and 2 without colostomy. All 6 had received a full course of chemotherapy with 5-fluorouracil (5-FU) and a variety of other chemotherapeutics. Two of the patients received radiation therapy. The response of these patients to the LSU treatment program was not as impressive as for instance, in the case of liver cancer patients. Only the 2 patients without colostomy survived more than five years after 2 and 3 LSU treatment courses respectively. In both cases, the CEA was maintained below 5.0 ng/mL. One of the colostomy patients died from a heart attack after a good initial response to the treatment in the 11th week of his treatment. 5-FU-induced myocardial ischemia may have been a contributing factor. Another of the colostomy patients apparently died from a barbiturate overdose, possibly a suicide attempt. It should be noted that over 35 of the colostomy patients were lost in the follow-up. The two patients who had suffered abdominal radiation had severe problems with adhesions and fistulas. Both had severe diarrhea at admission that was controlled with diet within about 2 to 3 weeks. Though both had a life expectancy of less than 3 months at the time of admission, they survived for 2 years and 7 months, and 3 years and 3 months, respectively. Their CEA levels returned to below 5.0 ng/mL after 3 months and stayed there until their deaths. 8. Uterine Cervical Cancers All 6 patients in this study (of a total of>30) had undergone radical hysterectomies and pelvic lymphadenectomies, multiple radiation treatments, and full courses of chemotherapy (4 patients received a combination of doxorubicin and methotrexate; 4 patients received mitomycine, vincristine, and bleomycin; one patient had been given both combinations). One patient died after 2 years and 20 months after undergoing 4 courses of the LSU treatment. Originally she was given less than 3 months to live. One patient fell down a flight of stars, fractured her neck and died with hours. She had survived 3 years and 5 months. Her original life expectancy was less than one year. Two patients survived 5 years and had no indication of tumor recurrence on CAT scans and NMR imaging. For the normalization of abnormal Papanicolaou (PAP) smears [Group 2: Infections; Group 4: squamous cell CA; Group 5: adenocarcinoma; Group 6: nonepithelial malignancy] and even of Stage O (Carcinoma in situ) through Stage IA2 (strictly confined to cervix; depth: £5 mm, spread: £7 mm), cervical cancers, topical application of folic acid in conjunction with vaginal ozone application has been found virtually 100% effectivein about 30 patients. Vaginal ozone applications are also an effective prevention of cervical cancers since it removes HPV and other pathogens that are causing chronic cervicitis that may turn malignant. 9. Brain Cancers All 4 brain cancer patients (of a total of about 15) had highly malignant extensive glioblastomas. All 4 had undergone surgery and radiation, as well as glucocorticoid therapy. Two of the patients were unconscious at admission. The two conscious patients complained about headaches, especially in the morning, loss of appetite, nausea, loss of concentration, reduced mental capacity, and increased sleepiness. In both, personality changes were clearly evident.

After treatment onset both unconscious patients regained consciousness within 3 days and were able to say simple sentences within 5 and 8 days respectively. One of these patients suddenly deteriorated in the 4th week, possibly from malnutrition. His sister, who supervised his feeding, had failed to properly follow our instructions. When we found out that there was a problem, the patient was already beyond recovery. The other patient recovered well enough to return to his job as a real estate broker, and has survived 5 years without sign of recurrence. Both of the two conscious patients had a lethal car accident; one about 2-1/2 years, the other about 3-1/2 years after their treatments. Both accidents may have been related to personality and psychomotor changes caused by their original tumors. 10. Melanomas The three patients with melanoma in this study (of a total of about 12) all had widespread metastatic disease. They all responded well to the first course of treatment though less favorably to further treatment courses. One of the patients died after 11 months. She had originally been given less than one month to live. Another patient who had been given less than 6 months to live survived 2 years and 10 months. One of the patients, a black woman who had undergone 5 courses of treatment, survived 5 years without sign of malignancy. 11. Other Cancers The number of the 10 remaining tumors in this study (of a total remaining of >80), two ovarian cancers, two stomach cancers (one of which was combined with a pancreatic cancer; see under 4.), one osteosarcoma, one soft tissue sarcoma, two kidney cancers, one bladder cancer, and one adrenal cancer, is too small to allow any clear judgment of the effectiveness of the LSU treatment in these specific cancers. In all cases, a prompt response was seen in the first treatment course. One kidney cancer patient died after 20 days as a consequence of his chemotherapy. The other kidney cancer patient responded moderately well to the LSU treatment and died after 4 years and one month (well over 5 years after his original diagnosis & thus “cured” according to NCI statistics,). The stomach cancer patient who also had pancreas cancer is described above under 4. He died after 1 year and 3 months. The other-stomach cancer patient responded moderately well to consecutive LSU treatments and died after 4 years and 2 months (rather than after less than one year ; & would also be listed by NCI as “cured”). One ovarian cancer patient responded well and survived over 5 years. The other responded moderately well to consecutive LSU treatments and survived 3 years and 10 months.The bladder cancer patient did not respond well and died after 11-1/2 months (rather than after less than 1 month). The adrenal cancer did well, needed only one LSU treatment course, and survived over 5 years without sign of recurrence. Continued next page

The 200 Plus Cancers Treated from 1987 through 2003 The following are only general remarks since on 2 May 1992, U.S Government Agents simultaneously broke into three locations where the originals and two copies of some 3000 patient records treated by LSU from 1980 through 1992, including about 650 cancer patients, about 180 AIDS patients, about 80 multiple sclerosis patients, and over 2000 patients with different conditions that were the data basis for the 2d ed. of the Ozone Book that for reasons beyond the control of the authors took until the year 2004 to be finally completed. . Again, we see a prevalence of “incurable” cancers (a) which have 0.0% success rate and thus should NOT be treated conventionally at all, including, small cell lung, pancreas, & esophagus cancers, acute adult leukemias, and all cancers with widespread metastasis; (b) malignancies where conventional treatment in almost all cases shortens the life span, including, stomach, brain, liver, & most ovarian cancers, multiple myeloma & chronic adult leukemias, as well as large (>10 cm = >2″) fast growing cancers with lymph node metastasis; (c) cancers with the highest incidence (in the USA & Western Europe), including, (female) breast, prostate, lungs[see (a)], & colon, where with early detection there is about 50% 5-year survival in breast, of 60% in prostate, & about 25% in colon cancers, that drops precipitously to some 10% if (b) & 1.0% if (a), supra, conditions are present; (d) other cancers including non-Hodgkin lymphomas, cancers of the urinary bladder & kidneys, rectum, (epi/naso)pharynx & oral cavity, endometrium & uterine cervix, & melanomas of the skin, rectovaginal cancer, larynx & thyroid cancer, Ewing sarcoma, etc. [which includes all 20 most frequent cancers in Thailand]. The estimated overall 5-year survival rate of all of these cancer patients, almost all of them terminal with widespread metastasis [see (a)] & [seeking our treatment only] after all conventional treatments had been exhausted, was ~40%, which increased to ~50% if they survived the first 3 weeks after treatment onset, & to ~60% if they survived 3 months after treatment onset, even more, ~80%, if they had a chance to have follow-up treatments at LSU, which was denied to virtually all patients after 17 July 1998 & until mid-2003, and many of which would be alive today; and while the estimated 5 year survival of untreated [with conventional methods: surgery &/or radiation &/or chemotherapy, etc.] patients was about 95% if they kept in touch with LSU/ULS, had a purpose to their lives with goals they absolutely needed to achieve, no matter what, meticulously maintained their alkalinizing blood-type-specific supplementation/diet/lifestyle, & balanced mind/ body/spirit as practitioners of Taoist Energy Healing, Silva Mind Control, & Neurolinguistic Programming (NLP).

Why is it essential that you stay in touch with us after completion of your initial treatment? Because we will use EVERY METHOD AVAILABLE to get & keep you well These methods, individually tailored to your specific needs, may include but are not limited to the following:

(both for treatment & maintenance): The most effective herbal electron donors that restore the body to an alkaline balance can be found in plants containing high amounts of germanium (Ge). Medicinal plants that reputedly have anticancer activity and that contain high amounts of Ge include shelf fungus (Trametes cinnabarina; 800- 2000 ppm), Ginseng (Panax ginseng; 250-350 Korean < 4000ppm), garlic (Allium sativum; 750 ppm), d?ng-sh?n/sansukon root (Codonopsis pilosula; 260 ppm), sushi (Angelica pubescens; 260 ppm), Bandai moss (260 ppm), Japanese waternut (Trapa japonica: 240 ppm), Comfrey (Symphytum officinale; 150 ppm), boxthorn seed (Lycium chinense; 125 ppm), wisteria knob/gall (Wisteria floribunda; 110 ppm), pearl barley (fructus coicis lacryma-jobi; 75 ppm), etc. Based on this concept, Kazuhiko Asai synthesized numerous non-toxic Ge compounds, most notably, propagermanium or biscarboxyethyl Ge sesquioxide [O3(Ge.CH2.CH2.COOH)2], which has been found effective in the prevention and treatment of numerous cancers and their metastases including cancers of the lungs, prostate, breast, liver, kidney, brain tumors, lymphomas and leukemias, and sarcomas such as chondro- and osteosarcomas. The recommended dosage for prevention is 100 to 200 mg/day and for treatment 1000 to 4000 mg/day for a 60 kg patient. Except for a Herxheimer-type “healing crisis” reaction, no other adverse effects have been observed with this compound. If no effect is seen, the treatment should be discontinued after 60 days.

Herbal treatments of cancer which were used worldwide since time immemorial include: Shark cartilage, Resistocell®, the thymus preparations Thymex L® and TFZ-Thymomodulin®, colostrum-derived transfer factor (TF) according to H. Hugh Fudenberg, Dr. Nieper¹s natural anticancer substances, and herbal cancer treatments such as compounded Hoksey [Trifolium pratense, Rhammus cathartica, Berberis vulgaris, Arctium lappa, Stillingia sylvatica, Rhammus purshiana or Cascara amarga (Sweetia panamensis), Glycyrrhiza glabra, Zanthoxylum clava-herculis], compounded Echinacea [Echinacea spp, Ceanothus americanus, Baptisia tinctoria, Thuja occidentalis, Stillingia sylvatica, Iris versicolor, Zanthoxylum clava-herculis], Folia Thujae occidentalis (fresh), Radix Astragali membranacei (Huáng Qí), Radix Rumicis crispi (fresh), and Renèe Caisse’s Essiac compound [Rumex acetosella, Arctium lappa (fresh root), Ulmus rubra, Rheum palmatum (root), etc.], PDR Cancer Formula [Larrea divaricata (folia), Sanguinaria canadensis (radix), Trifolium pratense (flores), Arcticum lappa (radix); Echinacea purpurea (radix), Hydrastis canadensis (radix); Symphytum officinale (folia), Eleutherococcus senticosus (radix; eventually folia, radix, and flores), Chelidonium maius, combined with Artemisia absinthium, Yucca spp, and Commiphora molmol (gum), C. abyssinica (myrrh), or C. opobalsamum (bdellium-oleoresin)], Laetrile® et al. mandelonitriles, immunostimulating mushroom extracts from Grifola frondosa (maitake), Ganoderma lucidum (reishi), and Lentinusedodes (shiitake), combined with herbs for specific cancers; e.g., herba Hedyotis diffusae (bái hu? shé c?o) combined with herba Scutellariae barbatae (bàn zh? lían) for stomach, esophageal, & colon cancers , & the latter alone for lung cancers, & tuber Dioscoreae bulbiferae (huáng yào z?) for thyroid cancer & endemic goiter, and, especially, Haelan 851® Platinum Formula and Natures Blessing.

What virtually all cancer survivors, particularly the ones that had been undergoing conventional therapies, have in common is that they had a purpose to their lives with goals they absolutely needed to achieve, no matter what. If counseling is successful in restructuring an individual’s outlook on life along those lines considerable life extensions beyond all expectations can be achieved after conventional therapies, while with the enhanced high pH therapy, the success is virtually guaranteed, provided that the patient has survived the first three months after the treatment started, and that they followed the programs outlined under 4. Conventional cancer treatment attempts, particularly surgery, that may in many cases frustrate all efforts to restore the will to live include colostomies, crippling lung resections, amputations of limbs, especially in children, cosmetically poor results after head, neck, & breast surgery &/or radiation. The same applies to paralysis after collapse of vertebrae from metastasis or from brain malignancies. Continued next page

Meticulously maintaining their prescribed alkalinizing blood type specific diet, supplementation, exercise program, and lifestyle is as essential as mental reconditioning [see 3.] and energy balancing [see 5.]. Individualized supplementation may include maintenance doses of cesium & rubidium, potassium & magnesium salts, Wobemugos, bromelain, papain, superoxide dismutase (SOD), & other enzymes, coenzyme Q10, vitamin A & beta-carotene, selenium & vitamin E, vitamin C, quercetin, & isoflavones, lycopene, N-acetyl cystein (NAC), pycnogenol, d-limonene, curcumin, alpha lipoic acid, inositol, methylsulfonylmethane (MSM), ellagic acid & graviola (Annona muricata), Primal Defense, Nature’s Blessing, green tea, olive leaf extract, echinacea, garlic, parsley, Korean ginseng, apricot pits, wheat grass, chlorella, cod & shark liver oils, contortrostatin, carrot & cabbage juices, mogu (Kompucha) tea, regular escargots & soy bean products for blood type As & ABs, and over 20 other cancer fighting foods according to your blood type & individually tailored to specific needs. The blood type specific diet & exercise program follows largely the one outlined in Dr. Peter J. D’Adamo’s book “Live Right Four Your Type”, modified & amplified based on our own research including avoidance of sugar & fructose ( & all refined carbohydrates) by all types, particularly Os & Bs, avoidance of cow’s milk, particularly Os & As, avoidance of the foods shown harmful for all types including pork, etc. All these programs have been streamlined and are available through people I have trained and shown a dedication to the ongoing development of High PH Therapy. With the most well structured program being available through Paul Rana of The RANA System in Australia, Dr Pablo at XYZ-Wellbeing Retreat Facility and Dr Sherrie in India.

 

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Abdul-Haqq H.E. Sartori, M.D

Page 4 of 5

RE: Enhanced High-pH Therapy for Cancer now available through trained Practitioner at XYZ Wellbeing ReTreat Facility founded in the year 2000 and undergoing a major refit and expansion in late 2008.

Thank you for contacting me to enquire about Cesium chloride (CsCl) and the Enhanced High-pH Therapy for Cancer originated by A. Keith Brewer, Ph.D., and since 1980 enhanced and perfected by myself.

Though the results were published in a major peer-reviewed medical journal, Pharmacology, Biochemistry, and Behavior in the December 1984 Supplement I, there was, except for the late Dr. Hans Nieper, a minimum of response from both the orthodox and alternative medical community.

Therefore, unfortunately, I am the only physician left who uses this by far most consistently effective therapy for all fast-growing cancers that have been treated so far, no matter what stage or type or extent. So as I am aging, I have trained a few people the correct and safe way to use this therapy. Do not be experimented on, my many years of research are beyond reproach.

Please read all my notes before you undertake any program. Since 1980, over 700 cancer patients have been treated with this therapy. In all cases, fast-growing tumors were promptly reduced in size with minimum discomfort to the patient (as compared to the common and sometimes horrendous adverse effects of chemotherapy and after radiation). With the intravenous (I.V.) application of this therapy, we consistently achieved primary & metastatic tumor reductions of 1.0 to 2.0 cm (2/5 to 2/5 of an inch) per day, i.e., disappearance of 5.0 cm (2.0) tumors in about four days, and of 10.0 cm (4.0) tumors in about eight days, and reductions of lymph node metastases of 2-5 mm/day.

Besides the higher and more consistent effectiveness, I.V. application of CsCl and other minerals, vitamins, mandelonitriles (e.g., Laetrile®), etc., avoids all side effects from oral therapy such as nausea, vomiting, diarrhea, abdominal discomfort, etc. Furthermore, I.V. application guarantees that all ingredients are taken up by the body, as often nutrient absorption may be compromised, particularly in patients with any type of malabsorption from gastrointestinal problems or in many advanced cancers or simply from lack of hydrochloric acid.

The only side effects seen with this therapy is the sometimes considerable, but brief, discomfort from the I.V. application of Ozone that is, in fact, a most beneficial homeopathic-type healing crisis. Best of all, this healing crisis reverses virtually all tendencies towards any type of illness and, in due time, almost all patients report that have “never felt better” in their entire life. In a tireless effort, Paul Rana, since 1998, developed most effective and comprehensive system in preparation for and as follow up of the Enhanced High pH Therapy.

The Rana System is an integral part of our therapy and you should follow it for at least one year or, preferably, for the rest of your life. Following this System gives you not only the highest success rates in permanently overcoming cancer but also greatly enhances your overall health, happiness, vigor, and longevity. For more information about The RANA SYSTEM and how to become a member, please consult with Paul Rana or peruse his websites in Australia.

I have passed on The RANA System research papers and system manuals with permission to www.xyz-wellbeing.com team 1995, early results are exciting to say the very least and the upgrade of a ReTreat Facility in Colombia is scheduled in 2008. Contact Dr Pablo at xyz for details.The Enhanced High-pH Therapy for cancer within the framework of The XYZ Wellbeing ReTreat System is now available in Colombia at a fraction of the financial costs of any conventional therapy that, besides very poor results in most cancers, causes severe suffering and in many cases permanent damages, and is the main cause for premature deaths in cancer patients. Since 1970, the start of President Nixon’s War on Cancer, the yearly death rate in the U.S.A. went up from 135,000 to over 800,000 and the average cost per patient is around US$ 300,000.00 ($ 100,000 to over $ 1,000,000.00) with an average out of pocket expenses for insured patients of about $ 60,000.00 ($ 20k to >200k).

Compared with this, the total all-inclusive investment for six to nine weeks of treatment in Colombia including the Enhanced High-pH Therapy for cancer (with room & board for a companion) and ongoing follow-up, as well as setup & three months of all supplements. They have designed a three month in house and 3 month follow up program that is under research that includes the best combination of services and the most determined team I have seen. If you are one of those patients that seek us out first when their primary tumor is less than 5.0 cm (2.0″) in diameter (and which have not yet undergone any conventional treatment), they should offer you a special price.

Also enclosed are my letters o

Endomatrial Cancer
Endometrium is the inner lining of the mammalian uterus and very susceptible hormone change, particular to menstrual cycle. Endometrial cancer is a late adulthood cancer defined as a condition of which the cells of the endometrial lining of uterus have growth uncontrollable or become cancerous as a result of the alternation of cells DNA. It’s the fourth most common cancer among women overall, after breast cancer, lung cancer, and bowel cancer.

Causes and risk factors
1. Age
Most endometrial cancer occurs to women in mid thirty and older. It may be caused by often than other tissue in growth and repair of the uterus lining over decade or many decades.

2. Estrogen
Because of accumulation and exposure of estrogen hormone over a prolonged period over a life time, risk of endometrial cancer is much higher in older women. In fact risk of endometrial cancer increase with age after age of 40. Women who are under age of 40 have low risk of the disease unless under usually risk factors.

3. Hormone replacement therapy (HRT)
Women who is either with ovaries removed or in stage of menopause use hormone replacement therapy to relieve the symptoms of menopause, such as frequency of hot flash, loss of bone density, mineral deficiency, etc may slightly increase the risk of endometrial cancer due to exposure to synthesis estrogen.

4. Oral contraceptive pills
The study used population-based cancer registries in eight geographic regions across the United States showed that women who had used sequential oral contraceptives (estrogen and progestin components taken at different times of the month) appeared to have an increased risk of endometrial cancer.

5. Tamoxifen
Tamoxifen is a common chemotherapy medication for patients with breast cancer or to prevent the recurrence of breast cancer by blocking the action of estrogen on the breast cancer cells. might increase risk of endometrial cancer as tamoxifen is also acts like estrogen itself.

6. Obesity
Fatty tissue is a good source for the production of bad estrogen in large amounts in obese women, leading to high levels of estrogen in women resulting in increasing risk of endometrial cancer.

7. Heredity
Family history of endometrial, colon and breast cancer increases the risk of endometrial cancer.

8. Abnormal overgrowth of the endometrium
Women diagnosed with endometrial hyperplasia (abnormal overgrowth of cells in the lining of the uterus) have a higher risk of developing endometrial cancer.

9. Infertility
Study found that women who have never been pregnant have a slightly higher risk to develop endometrial cancer.

10. Early puberty before age 12
Women who begin their periods before 12 years of age have a higher risk to develop endometrial cancer due to increased the number of years that the immature inner uterine lining is exposed to estrogen.

11. Menopause after age 55
Women who go through Menopause after age 55 are at a higher risk of developing endometrial cancer because of increased the number of years that the inner uterine lining is exposed to estrogen.

12. History of having radiation therapy to the pelvis
Frequent use of radiation therapy to the pelvic may alter and damage the DNA of cells, leading to uncontrollable cells growth of inner lining of the uterus, causing endometrial cancer.

13. Other reproductive cancers
Breast or ovary cancer are associated with an increased risk of endometrial cancer due exposure to higher than normal amount of estrogen.

14. Intake high amount of fat
Intake high amount of fat is also linked to higher amount of estrogen produced by our body, the “good” source hormone for the development of endometrial cancer.

15. Heavy daily alcohol consumption
researcher found that high alcohol consumption not only affect just the liver, but increases the risk of with breast and endometrial cancer.

16. Etc.
Symptoms
Abnormal uterine bleeding may be the early stage of endometrial cancer
1. Abnormal vaginal bleeding
Vaginal bleeding between period with in inappropriate amounts of blood flow should be checked with doctor as soon as possible. It may be caused by expansion of over growth of pre cancer uterine lining cells causes of infection or breaking off the endometrial capillaries or blood vessels.

2. Increase amount of vaginal discharge
An increase in the amount of vaginal discharge accompanied with abnormal odor or pain, itching, or burning sensation may be caused by infection or inflammation.

3. Painful urination
Painful urination, most of the time is caused by infection of the urinary tract that affect the bladder in emptying, but sometime it can be caused by infectious diseases, and may be an indication of pre endometrial cancer.

4. Pain during sex
Painful during most of the time is caused by vestibulitus and vaginismus sex, but in some cases, it can be caused by cancer or other reproductive diseases.

5. Pain in the abdomen
There are many reproductive disease can cause pain in the pelvic, such as appendicitis, salpingitis, uterine descensus, etc. but it can be caused by early stage of endometrial cancer.

6. Swelling or Lump in the pelvic
Swelling can be caused by inflammation of sexual transmitting virus or infection by bacteria. Lump may be caused by abnormal cells growth of the uterine lining or reproductive tissue.

7. Weight loss
Unintentional weight loss due to loss of appetite may be a first sign of cancers, including uterine inner limning.

9. Anemia
Anemia may be caused by chronic loss of blood due to prolonged or heavy abnormal menstrual bleeding.

10. Etc.

If you have any of above symptom and accompanied with abnormal uterine bleeding, the diagnosis beside required a detail of you health history, including family medical history, menstrual and pregnancy history, lifestyle, etc.
1. Pelvic examination
Pelvic examination taken when there is no menstruation and advised not to have sex for at least 24 hours, is type of complete physical exam of a woman pelvic organ by a medical instrument to detect any infection, abnormal cells growth such as cysts, fibroid and specially for virus which can cause early stage of endometrial cancer. The pap test usually is also taken. If your doctor found any abnormal cells growth or suspected uterine cancer, further tests may be required.

2. Endometrial curettage
Ednometrial curettage can be done in the doctor office or in the hospital as an outpatient by taking a sample of tissue from the inner lining layer of the uterus. In some cases, if the example taken by endometrial curettage does not review a sufficient diagnostic result, a dilation and curettage (D&C) is necessary.

3. Dilation and curettage (D&C)
Dilation and curettage is a types of examination by gently opening the cervix and then removing some of the inner lining (endometrium) of the uterine cells. During the test, you doctor may remove any endometrial polyps, if found.

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The 2. and 3. are operated under local anesthesia with anti-inflammatory and pain medication before and during examination.

4. Hysteroscopy
Hysteroscopy is the pelvic examination by the use of an endoscope that carries optical and light channels or fibers that go through the opening of the vagina then into uterus and allows your doctor can see the uterine inner lining (endometrium) on a video screen with the help of fluids or CO2 gas is introduced to expand the cavity during the examination. A small sample is taken for viewing under microscope.

5. Endometrial biopsy
Endometrial biopsy or aspiration is another procedure to remove a sample from the uterine inner lining with the inserting of biopsy curettage into the uterine and with a scraping and rotating motion. During this procedure
a. A speculum helps to spread the walls of the vagina apart to expose the cervix.
b. A tenaculum helps to hold the cervix steady.
c. Local anesthesia may or may not be given.

6. Trans-vaginal ultrasound
Trans-vaginal ultrasound is a type of pelvic ultrasound used to diagnose the thickness of the uterine inner lining of the women with post-menopause bleeding for endometrial cancer with the help of a transducer (The transducer is necessary for sending out sound waves, which reflect off body structures), into the vagina.

7. Sonohysterography
In some cases, more clearly view the uterus is necessary to get a better view of the size of tumor, then sonohysterography is used with saline (sterile salt water) injected into the uterus before the ultrasound.

8. Computerized tomography (CT) scan
CT scanning combines special x-ray equipment with sophisticated computers to produce multiple cross-sectional images of any susceptible area of the inner uterus lining and surrounded organs if need, to the computer screen around a single axis of rotation.

9. Magnetic resonance imaging (MRI)
MRI uses a powerful magnetic and radio frequency field to capture 3D image of organs, soft tissues, bone and virtually all other internal body structures, including endometrial cancer to the computer screen for examination.

10. Etc.

A. Adenocarcinomas
Adenocarcinomas is accounted for accounts for over 80% of cases of endometrial cancer and begun with the abnormal cells growth in the surface cells of the inner lining of the Researcher found that women who have an early onset of symptoms of peri-menopause are in higher risk of adenocarcinomas.

B. Uterine sarcoma
Uterine sarcoma,a rare uterine cancer containing malignant cells of the muscle and connective tissue of the uterus, is accounted for only account for only 2 percent of all uterine cancer.
a.Endometrial stromal sarcomas
Endometrial stromal sarcomas are also endometrial cancers originated in the non-glandular connective tissue of the endometrium, therefore the cell of origin is unknown.

b. Leiomyosarcomas
Leiomyosarcomas is a form of endometrial cancer originated from the smooth muscles of the uterus. The cancer has a resistant tendency and not very responsive to chemotherapy or radiation.

c. Carcinosarcoma
Carcinosarcoma is a type of cancer caused by both epithelial and connective tissues of the uterus.

E. Etc.

Grades of endometrial cancer
1. Type 1 endometrial cancers
endometrial cancers are caused by excess estrogen. These types of cancer is not very aggressive and are slow to spread to other tissues and considered as low grade.
2. Type 2 endometrial cancer
Endometrial cancer not caused by excess estrogen. These types of cancer are invasive and have a tendency to spread to distant parts of the body and considered high grade.

Stages and grade of endometrial cancer is important to determine the best treatment with the aim is to cure and prevent recurrence, if that it is possible.
1. Stages 0
Cancer is found in the surface of the endometrial lining.
2. Stage I
Cancer have penetrated into the endometrial lining but within the uterus.
a. Stage IA
Cancer is limited to the endometrium
b. Stage IB:
cancer have penetrated to middle layer of the endometrial linning wall
Stage II
Cancer is now present in both the uterus and cervix.
Stage III
Cancer has spread beyond the uterus and cervix to the lymph nodes, but hasn’t reached the rectum and bladder.
a. Stage IIIA:
Cnacer has spread beyond outermost layer of the uterus and to the ovaries or fallopian tubes
b. Stage IIIB:
Cancer has spread to the vagina and to the lymph node area
c. Stage IIIC
Cancer has spread to the lymph nodes but not to distant organs
Stage IV
Cancer has spread to distant parts of the body
a. Stage IVA:
Cnacer has spread to rectum and bladder
b. Stage IVB
Cancer has spread to distant organs of the body.
Prevention
1. Maintain a healthy weight
Obesity increases the risk of endometrial cancer.
2. Reduce intake of alcohol
Researcher found that association of alcohol intake and endometrial cancer is stronger among lean women than among overweight or obese postmenopausal women.
3. Birth-control pills
Birth control bill is found to reduce the risk of endometrial cancer
4. Avoid unopposed estrogen therapy
Excess estrogen increases the risk of endometrial cancer.
5. Antioxidants
Antioxidants is found to reduce the risk of cancer caused by oxidation, inducing free radical scavenging such vitamin A, C, E and others.
6. Enhance immune system
By getting enough sleep, eating healthy diet, exercise and living healthy lifestyle.
7. Vitamin D
Vitamin D has been shown to be helpful in a number of cancers, it may also good for endometrial cancer.
8. Etc.

Treatments
Depending to the grade and stage of the cancer
1. Surgery
Most patients with invasive or non invasive cervical cancer are required surgery. While non invasive cancer patients are not needed further treatment, invasive cancer patients usual needed further treatments, including radiation therapy, chemotherapy. In some cases, example of lymph node is also remove, ovaries and uterus also removed, depending to the surgery needed.

2. Radiotherapy
Radiation may be used for stages II, III, and IV and usually given after surgery to kill any cancer cells remaining in the body. By using high-energy x-rays or other types of radiation, radiation therapy kills endometrial cancer cells and keeps them from growing or regrowing. If the cancer is small in size, cancer can be cured. If the cancer is large, radiation therapy can be used to control local bad symptoms.
a. External beam irradiation
In External beam therapy (EBT), a beam of high-energy x-rays or or other types of radiation is directed to a patient’s tumor externally depending to the stage of the cancer.
b. Brachytherapy
By placing a small quantity radioactive seeds or sources by a medical instrument into uterus or vagina, before using high-energy x-rays or other types of radiation to kill cancerous cells without causing radiation affects in the nearby healthy tissues.
c. Side effects
c.1. Fatigue
c.2. Chest pain
c.3. Heart problem
c.4. Short of breath
c.5. Skin discoloration or pinkness, irritation.
c.6. Etc.

3. Chemotherapy
a. Chemotherapy is most use to treat with advance stage of cervical cancer combined with radio therapy, as it has spread to a distant parts of the body by using drugs taken by mouth or injected into a vein or muscle of the patient to stop the growth of or to kill cancer cells.
b. Side effects
b.1. Nausea
b.2. Vomiting
b.3. Hair loss
b.4. Fatigue
b.5. Anemia
b.6. Mouth sores taste and smell changes
b.7. Infection
b.8. Etc.

4. Hormone therapy
Hormone therapy is used in the pre-endomatrial cancer stage or where surgery is either feasible or unnecessary. Use of right types of hormone can cause the shed of endometrial lining or endometrial cancer to shrink or sometimes disappear completely.

Other unconventional treatments
A. With herbs
1. Trifolium pratense
Trifolium pratense is best known as Red clover, a genus Trifolium, belongs to the familyFabaceae, native to Europe, Western Asia and northwest Africa. Researcher found that genistein inhibits of cancer cell growth, promotes apoptosis.

2. Trifolium pratense
Trifolium pratense is best known as Red clover, a genus Trifolium, belongs to the familyFabaceae, native to Europe, Western Asia and northwest Africa. Researcher found that genistein inhibits of cancer cell growth, promotes apoptosis.

3. Soy isoflavone distillate
Soy isoflavone distillate is a extract of the beans of the soy plant or herb, it contains high amount of phytoestrogen by binding to estrogen in the human cells. Researcher found that soy isoflavones wards off estrogen from the cells which are receptive to estrogen, including the cancerous or malignant cells endometrium. There is some conflicts in the studies of the use of phytoestrogen in treating estrogen related cancer, please be careful not to use them unless with the suggestion of your specialist in the related field

4. Astragalus
Astragalus is herb, genus of Astragalus, belonging to the legume family Fabaceae. In rat study, researcher found that astragalus extract possess cytostatic properties in inhibiting tumor growth and delay chemical-induced hepatocarcinogenesis.

5. The inner bark of Cat’s claw
The herb has been used in herbal medicine to boost function of the immune system and treat and prevent infection. In vivo and in vitro, cat’s claw showed inflammation inhibiting effect. It also contains vary chemicals such as tannins, catechins, and procyanidins which demanstrated antioxidant and anti-inflammatory properties.

6. Etc.

B. With Chinese herbs
1. Xia Ku Cao
Xia Ku Cao is also known as Self heal plant. The acrid, bitter and cold herb has been used in TCM to treat headache, dizziness, tuberculosis of the lungs, vaginal infection, excessive bleeding during menopause as it clears liver and heat, dissipates nodules and dissolves phlegm by enhacing the functions of liver and gallbladder channels.
Researcher found that Xia Ku Cao significantly reduced the endometrial cancer cells growth, in mice implanted with human endometrial cancer cells and demonstrated the anti-estrogen effect in the study as well.

2. Dong Ling Cao
Gong ling Cao is also known as blushred rabdosia. The sweet, bitter and cold herb has been used in TCM to treat chronic bronchitis, chronic inflammation of the pelvic area, swelling of throat, insect bites and snake bites as it clears heat and toxins, nourish yin, remove blood stasis and relieves pain by enhancing the functions of stomach, lung and liver channels. In vitro, researcher found that Gong ling Cao has cytotxicity activity against CaEs-17 human esophageal cancer cell line, at 2 and 3ug/ml, the inhibiting rates were 40% and 75%. Can it be used effective against endometrial cancer? Further studies is needed.

3. Lu Feng Fang
Lu feng Fang is also known as Honeycomb. The sweet, acrid, neutral and toxic herb has been used in TCM as diuretic and to strengthen the heart activity, lower blood pressure temporary, promote the coagulation of blood, treat of cancer, bleeding in tumors as it relieves toxicity, expels wind, alleviates pain, dries dampness by enhancing the functions of lung, liver, spleen, stomach and large intestine channels. In vitro experiments researchers found that lu feng fang demonstrate the inhibition of human liver cancer cells. In insect study by using HeLa cells originating from human cervix and uterine cancer, researchers also found that Lu Feng Fang extract showed a potent anticancer activity.

4. Bai Hua She She Cao
Bai Hua She She Cao is also known as spreading hedyotis, The bitter, sweet and cold herb has been used in TCM as anti-bacteria, anti-inflammation, anti-tumor, anti-virus agent and to treat snakebite and enhances immune system as it clears heat, drains dampness, expels toxins and resolves abscesses by enhancing the functions of liver, stomach, large intestine channels.
The Sanjiv Kumar YADAV, Shao Chin LEE(Yong Loo Lin School of Medicine, National University of Singapore researcher results showed that the ethanol extract from Bai Hua She She Cao effectively evokes cancer cell apoptosis, possibly through burst-mediated caspase activation.

5. Qing Hao
Qing Hao is also known as Worm Wood. The bitter and cold herb has been used in TCM as anti malaria agent and to trealupus, schistosomiasis as it disperses cold and dampness, expels wind and calms pain by enhancing the functions of kidney, liver and gallbladder channels.
Researchers found that qing hao elevates the calcium ions of leukemia white cells, triggering apoptosis, a self-destruct mechanism in cancer cells. A University of Washington study showed that qing hao selectively kills several cancer cell lines in the test tube.

6. Etc.

For other cancers or health articles, please visit http://medicaladvisorjournals.blogspot.com/

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The bladder is a hollow elastic organ in the center of the lower abdomen that collects urine from the kidneys and excreted them through the urethra.
Bladder cancer is most common type of cancer that effect men twice as frequently as it effects women. Usually it starts from the lining the bladder caused by several types of malignant growths of the urinary bladder cells.

Types of Bladder cancer
A. There are three types of bladder cancer beginning in cells in the lining of the bladder, classified due to their origination.
1. Transitional cell carcinoma
In early stage of bladder cancer, normal cells become cancerous in the innermost tissue layer of the bladder which can change shape and stretch without breaking apart as they are able to stretch when the bladder is full and shrink when it is emptied. More than 90 percent of bladder cancers begin in the transitional cells
2. Squamous cell carcinoma
Cancer begins in squamous cells in the found in the tissues of the surface of the bladder, due to long-term infection or irritation. About 8 percent of people with bladder cancers begin squamous cells
3. Adenocarcinoma
Cancer begins originates in glandular tissue of the bladder, including the surface layer of skin, glands and a variety of other tissue, due to long-term irritation and inflammation. Only 2 percent of people with the condition have a third bladder cancer type.

B. Types of bladder cancer classified due to their proliferation
1. Superficial types of bladder cancer
This type of bladder cancers have a characteristic of often recurrence but usually non invasive. It can be controlled or removed by regular cystoscopy.
2. Invasive types of bladder cancer
Invasive types of bladder cancer tend to spread after beginning in cells in the lining of the bladder. It often requires surgery by removing part or whole bladder.

Stages of bladder cancer
Bladder cancer can be classified in difference stage depending to the location, size, and spread of the cancer.Stage 0: Cancer cells are found only on the inner lining of the bladder Stage I: Cancer cells have spread to the layer beyond the inner lining of the bladder but not to the muscles.Stage II: Cancer cells have spread to the muscles in the bladder wall but not to the fatty tissue that surrounds the bladder.Stage III: Cancer cells have spread to the fatty tissue surrounding the urinary bladder and nearby organs, but not to the lymph nodes or other organs.Stage IV: Cancer cells have spread to the lymph nodes, and/or other organs, etc.Recurrent: Cancer has recurred in the bladder and/or another nearby organs after treatments.

Symptoms
1. Bladder spasms
2. Blood in urine
3. Frequent urination
4. Pain or burning sensation during urination
5. Pain in lower back and/or abdomen
6. Inability to urinate
7. Urinary urgency
8. Reduced bladder capacity
9. Difficult in urination
10. Loss of appetite and weight
11. Etc.

Causes
1. Smoking
Everybody knows that cigarette smoking is a major cause of lung cancer, but not many people understand that it also can lead to bladder cancer. Study of a group of people with the available smoking information, included duration of smoking habit, number of cigarettes smoked per day and time since cessation of smoking habit for ex-smokers, showed cigarette smoking increases the risk of bladder cancer with 1.96 after 20 years of smoking to 5.57 after 60 years, with an immediate decrease in risk of bladder cancer was observed for those who gave up smoking by over 30%. In fact, cigarette contains certain harmful chemicals which can also lead to bladder cancer due to prolong period of exposure in the limning of bladder walls.

2. Chemicals exposure
Reseachers investigated the risk of bladder cancer in association with exposure to over 12 000 occupational chemical agents, complex mixtures, and other substances, showed that increased risk of bladder cancer was detected for ever exposure to 635 chemical agents, and 341 chemical agents exhibited a significantly increasing dose-response relationship.

3. Diet high in fried meats and fat
Study showed that risk of bladder cancer increased by 37% with a high fat intake, by 40% with low fruit consumption and by 16% with diets low in vegetables.

4. Aging
The acceptance of that aging is one the greatest single risk factor for developing bladder cancer due to weakened immune system, increased free radicals frequent attacked, genetic and epigenetic changes, environmental influences, etc.

5. Infection caused by a certain parasite
A parasite worm, Schistosoma which can lead to Schistosomiasis, is a chronic disease that can damage a person internal organs and the urinary form of schistosomiasis may cause bladder cancer in adults. Schistosomiasis can also lead to inflammation of bladder lining leading to the transitional cell carcinoma, the predominant type of bladder cancer.

6. Etc.

Diagnosis
Diagnosis are always important for people who have had the symptoms mentioned above. If bladder cancer is diagnosed early, most of the times it can be cured.
Beside checking the patient personal health history and smoking habit, rectal examination for men and pelvic examination, others exams include
1. Urine test
Urine test is one of the first test which your doctor may order to rule out conditions of infection or inflammation causes of similar symptoms and detect the presence of certain antibodies and other markers may indicate bladder cancer.

2. Urinalysis:
Any abnormality found in urine test will be sent for urinalysis such as blood, protein, and sugar.

3. Cystoscopy
If you urine test came back positive, your doctor may want to take a look inside your bladder for abnormalities such as tumors by inserting a very narrow tube with a light and a camera on the end through the urethra. The camera transmits pictures to a video monitor for a direct viewing of the inside of the bladder wall.

4. Intravenous Pyelography (IVP)
The test is performed by taking a series of x-ray films of your bladder after injecting a drug solution with a catheter through the urethra to highlight and mark any abnormality inside your bladder easier. You are asked not to pass urine for a few hour after injection.

5. CT scan
CT scan can dive your doctor a three-dimensional view of your bladder and the rest of your urinary tract to look for masses and other abnormalities such as tumor.

6. Biopsy
This test usually is done under local anesthesia. If the lump is found in the bladder wall or area which is suspicious to be tumor, a tiny samples of your bladder wall are removed usually during cystoscopy and examined by a pathologist. Small tumors can also be removed during the biopsy process.

7. Etc.

Treatments
1. Surgery
a. Removal of superficial tumors
Normally if the tumor is the superficial type,general anesthesia is sometimes used for operative cystoscopic which is endoscopy of the urinary bladder via the urethra.

b. Total cystectomy
When cancer invades the muscle of the bladder wall, it is usually necessary to remove the entire bladder surgically, but for women patients, researchers advice that total cystectomy could manage invasive bladder cancer in women patients but night continence is not as good as that in men patients.

c. Partial cystectomy
Sometime by preserving the bladder, partial or segmental cystectomy is involved to remove only part of bladder tumor and surrounding bladder wall but researchers found that only 5.8-18.9% of patients with muscle-invasive bladder cancer are suitable candidates for partial cystectomy.

2. Radiotherapy
After the invasive cancer tumor is removed, if cancer cells are found on the edge of the removal tumor, your specialist may suggest radiotherapy to kill and prevent the cancer cells from spreading or if you want to try to keep your bladder, or try to keep your potency. Radiotherapy is the use of high energy rays to kill cancer cells. Treatments are usually 3 times a week, for up to 6 weeks or longer.

3. Chemotherapy
Chemotherapy is a method of treating cancer by using single drugs or combination of drugs to kill cancerous cells, especially for spreading quickly cancer cells or cancer cells have spread to other parts of body. In some case, chemotherapy may be recommended before surgery and/or radiotherapy.

4. Phototherapy
Phototherapy is treatment of a disease by exposing to light, especially by variously concentrated light rays or specific wavelengths. In one study, researchers found that the best results of treating bladder cancer by phototherapy were noted in tumors less than six mm. in diameter when treated with 15 mg./kg dihematoporphyrin ether (DHE) and exposed to 100 to 180 minutes of visible light.(Source)

Read more at http://www.wrongdiagnosis.com/treat/phototherapy.htm?ktrack=kcplinkPorphyrin-sensitized photoradiation therapy (PSPT) is a promising treatment for a variety of malignant tumors. Researchers found that phototherapy have a potential for a new modality in treating recurrent and resistant bladder cancer. Information gained form the basic studies will provide a better understanding for mechanisms of porphyrin uptake, retention and photosensitization in tumor cells, thus providing a basis for improving the treatment by increasing the efficacy and reducing the side effects. The development and the information can also be useful in the treatment of other tumors by PSPT.

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5. Intravesical or instillation
Bladder instillations are treatments of putting drugs directly into the bladder by inserting a catheter through the urethra, normally without using general anesthesia. You are asked not to urine for a few hours. The treatment sometimes cause temporary bleeding and bladder irritation.

6. Etc.

Nutritional Supplements
If you have read my previous article, Anti-Aging – How to Live Longer & Healthier:Antioxidants, Free Radicals, Cancers, Diseases, You might already know that vitamins and minerals play an important in preventing and treating DNA mutation in cells division and replication. In fact, they are vital for people with bladder cancer
1. Vitamin B6
Study found that Vitamin B6 in several clinical trials in bladder cancer, may benefit patients with bladder cancer by improving immune function fighting against the forming of free radical and irregular cells growth causes of bladder cancer.

2. Vitamin D
Vitamin D is best known for its function in nehance the absorption of calcium in preventing osteoporosis, but in bladder cancer, studies found that exposure to ultraviolet B (UVB) activates photosynthesis of vitamin D3 in elevated levels in the body, lowering the risk of bladder cancers.

3. Vitamin E
Regular use of vitamin E supplement for longer than 10 years was associated with a reduced risk of bladder cancer mortality , but regular use of shorter duration was not.

4. Selenium
‘The lower the levels of selenium, the higher the risk of developing bladder cancer,’ said lead researcher Nuria Malats, M.D., Ph.D., leader of the Genetic and Molecular Epidemiology Group, Human Cancer Genetics Program, Spanish National Cancer Research Centre, but the researchers noted a significant protective effect of selenium, mainly among women, which they believe may result from gender-specific differences in the mineral’s accumulation and excretion in women.

5. Carotenoids
Researchers from UT M.D. Anderson Cancer Center, Houston, TX, UT Health Science Center School of Public Health, Houston, TX, Baylor College of Medicine and Methodist Hospital, Houston, TX found that further evidence for a chemopreventive role for carotenoids by demonstrating that high carotenoid intake is associated with an overall decrease in BC risk and provided a reduction in risk for smokers and among individuals susceptible to DNA damage. These data may have important implications for cancer prevention, especially for individuals susceptible to DNA damage.

6. Combination of carotenoids, vitamin D , thiamin, niacin, and vitamin E
researchers found that in older individuals, the highest average intakes of the above were all associated with a reduced bladder cancer risk. In smokers, they also found that the highest intakes of vitamin E, carotenoids and niacin, were associated with a 42, 38, and 34 per cent reduction in bladder cancer risk in heavy smokers.

7. phosphorus
Some study found that phosphorus intakes also reduce the risk in bladder cancer.

Diet
1. calciferous vegetables
Studies found that increased intake of calciferous vegetables may slash the risk of bladder cancer by 36 per cent, says new research that attributes the benefits to the isothiocyanate content.
The Roswell Park researchers collected dietary data from 275 hospital-based bladder cancer patients and 825 cancer-free people, and found that those who consumed the highest consumption of raw cruciferous vegetables was associated with a 36 per cent reduction ibladder cancer risk

 

 

 

Furthermore, stronger protective effects were observed among current and heavy smokers with an intake of three or more servings of raw cruciferous vegetables per month associated with a 54 and 40 per cent reduction, respectively.

2. Soybeans
Reseachers found that genistein, soy phytochemical concentrate, and soy protein isolate have the ability to inhibit the growth of transplantable murine bladder cancer in vivo. Genistein, dietary soy phytochemical concentrate, and dietary soy protein isolate reduced tumor size by 40%, 48%, and 37%, respectively, as compared with controls.

3. Garlic
Chinese garlic-lovers are not the only ones to benefit from garlic’s cancer-fighting abilities, including bladder cancer. John Milner, Ph.d., from Pennsylvania State University, points out in a review article tracking the anti-cancer effects of garlic that “the potential benefits of garlic appear not to be limited to a specific region of the world …” when researchers noticed that a certain region of this country had both an extraordinarily high garlic intake and a surprisingly low rate of stomach cancer.

4. Green tea

Green tea is made from unfermented leaves and reportedly contains the highest concentration of powerful antioxidants called polyphenols the free radical scavengers that limit the damaging of DNA alternation resulting in lessening the risk of cancer, including bladder cancer. Green tea also has cancer-fighting properties and may cut off blood vessels that feed cancerous tumors.

Bladder cancer
Bladder cancer occurs when cells amass in the inner lining of the bladder and form a malignant growth, or tumor. Cancer develops only in the bladder lining may become invasive, spreading through the bladder wall to nearby organs such as the prostate gland in men or the uterus in women.

1. Traditional Chinese medicine indicated that urinary with blood is one of the first symptom discovered by bladder cancer patient. Sometimes, red blood cells can be viewed by naked eyes but most of the times, it required blood test.
2. Back pain
3. Fluid retention in the kidney
4. Fever
5. Other symptoms are similar to the conventional medicine.
Most patient with early bladder cancer my experience blood in urine with no pain at all.

1. Occupation
Occupation required to contact with chemical toxins daily and over a prolonged period of time have a high risk of bladder cancer. Most workers do not experience any symptom of bladder cancer until later in their life. We are taking about the period over 30 years.

2. Smoking
Cigarette contains certain harmful chemicals which can cause cancer. In China, due to social culture difference, the risk of male and female with bladder cancer is 41%, 30% accordingly.

3. Age
The risk of bladder cancer increases with age. The older you are, the higher the risk.

4. Sex
The risk of bladder cancer of male is 4 time higher than women

5. Race
Certain race are high risk of bladder cancer

6. Chemicals tryptophan
By products of tryptophan such as 3-hydroxy-2-amino-acetophenone and 3-Hydroxyindazole
can promote the risk of bladder cancer due to reaction of β-glucuronidases.

7. Diet
Diet high in saturated fat increases the risk of bladder cancer.

8. family history
Bladder cancer risk increase if a member of the direct family have bladder cancer.

9. Infection
Prolonged infection or frequent infection increase the risk of bladder cancer.

10. Medication
Certain types of medication can promote the forming of bladder cancer.

11. Etc.

The traditional Chinese medicine defined bladder cancer is a health condition caused by
A. Heart Fire Shifting Downwards to the small intestine
Heart Fire is an excess problem, due to elevating yang or yang rise is out of control. Since heart is associated with the fire, excessive yang leading to fire and heat. If heart fire with extra heat move downward to the small intestine, it affects the bladder in functioning of urinary secretion.
If left untreated, it can cause inflammation and bleeding, leading to bladder cancer.
1. Symptoms include
1.a. Heart palpitations
1.b. Thirst
1.c. Feeling of heat
1.d. Urination with blood
1.e. Hot, dark urine
1.f. Bitter taste in the mouth
1.g. Frequent urination
1.h. Urgent need to urinate
1.i. Etc.

2. Chinese Herbs
a. Shui Niu Jiao (Bubalus bubalis)
Shui Niu Jiao is also known as water buffalo horn. The salty, cold herb has been used in TCM as sedative, anti-inflammatory, anti-virus medicine and to shorten bleeding time, enhance quality of capillaries as it clears heat, relieve fire, cool blood, stop nosebleed, vomiting blood by enhancing the function of heart, liver, spleen, stomach channels.

b. Lian Qiao(Fructus Forsythiae Suspensae)
Lian Qiao is also known as forsythia fruit. The bitter, cool, and slightly acrid herb has been used in TCM as anti inflammatory medicine and to stop vomiting, promote blood circulation and urination as it helps to clears heat and toxin and expels externally wind heat by enhancing the function of heart, liver gall bladder channels.

c. Zhu Ye (Herba Loaphatheri Gracilis)
Zhu ye is also known as bamboo leaves. The sweet, cold and bitter herb has been used in TCM to
treat mouth sores and inflamed gums caused by heat, promotes urination and treat insomnia as it helps to clear damp heat, heart fire and stomach heat by enhancing the functions of heart, small intestine, stomach channels.

d. Lian Zi Xin (Lotus Plumule)
Lian Zi Xin also known as Lotus Flower. The bitter and cold herb has been used in TCM as an astringent and to treat insomnia, irritability, stop bleeding and vomiting blood as it promotes the draining of heart fire and bind essence by enhancing the functions of heart and pericardium channels.

e. Etc.

B. Damp heat moves downward to the bladder
It is caused by kidney inability to move fluid upward, causing fluid accumulated in the abdomen. Prolonged period of fluid accumulation promote arise of heat that affect the urinary secretion of the bladder, resulting of bleeding and damage the functioning of the bladder, leading to bladder cancer, if untreated for a prolonged period of time.
1. Symptoms include
1.a. Frequent urination
1.b. Urgent need to urinate
1.c. Urinary pain, back pain, edema of lower limbs
1.d. Thirsty,
1.e. Difficult to go to sleep at night
1.f. Red tongue with yellow and greasy fur
1.g. Slippery and taut pulse
1.h. Etc.

2. Chinese Herbs
The function of the herbs are for diuresis, to eliminate dampness, clear heat, cool blood and stop bleeding.
a. Che Qian Zi (Semen Plantaginis)
Che Qian Zi is also known as Plantain seed. The sweet and cold herb has been used in TCM as an antibiotic and to promote urination and to excret mucus of the bronchial tubes as it helps to clear Damp heat in the lower burner channel and damp heat cause of painful urinary, diarrhea , red eyes and sexual dysfunction by enhancing the functions of bladder, kidney, liver, lung channels.

b. Bian Xu (Herba Polygoni Avicularis)
Bian Xu is also known as common knotgrass. The slight cold and bitter herb has been used in TCM to treat urinary infection, get rid of skin parasites and fuild and worms and stop itching as it drains damp heat in bladder, calms pain on urination by enhancing the function of bladder channel.

c. Bo Ye (Cacumen Biotae Orientalis)
Bo Ye or Ce Bai Ye is also known as biota leaves. The bitter, tart and slightly cold herb has been used in TCM to stops bleeding, harmonize blood, inhibit cough, anti-bacteria and anti-inflammation as it purifies and lowers the lung-Qi , clears heat and blood heat, eliminates phlegm by enhancing the function of heart, liver, large intestine and lung channels.

d. Yi Yi Ren (Semen coicis)
Yi Yi Ren is also known as Coix Seed. The sweet, bland, slightly cool herb has been used in TCM to improve digestion, enhance the lung function, get rid of bacterial or fungal infection and treat neualgia, difficulty in urination, lung abscess as it drains water, strengthens the Spleen; eliminates obstruction and clears Heat by promoting the functions of spleen, stomach, lung channels.

e. Pu Gong Ying ( Herba Taraxaci Mongolici cum Radice)
Pu Gong Ying also best known as Dandelion. The bitter, cold and sweet herb has been used in TCM as a diuretic and to promote bile secretion, protect the liver, inhibit staphylococcus aureus, Streptococcus pneumoniae, Pseudomonas aeruginosa as it clears heat, expels toxins, drains Dampness by enhencing the function of liver and stomach channels.

f. Etc.

C. Kidney and liver yin deficiency
Kidney and liver yin deficiency cause of yang elevation, toxin accumulation and blood stagnation, leading to the forming of tumor in the bladder
This type is mostly seen at the advanced age, or chemotherapy or radiotherapy cause of heat reaction or complicated by infection.
1. Symptoms include
1.a. Frequent urination, sometimes
1.b. Blood in the urine
1. c. Abdominal distension and pain
1.d. Back pain
1.e. Dry mouth but don’t like to drink
1.f. Constipation
1.g. Light red tongue with little fur, or uncoated and smooth tongue
1. h. Thready pulse
1. i. Etc.

2. Herbs
The main functions of herb are to nourish yin, invigorate kidney, clear stagnation and detoxify.
a. Mai Dong (Tuber Ophiopogonis Japonici)
Mai Dong is also known as Ophiopogon Root. The sweet, slight bitter and cold herb has been used in TCM to improve contraction of heart muscles, recovery of cells of the pancreas and immune system, protect heart muscle, lower blood sugar and anti-arrhythmia as it
moistens the lungs, nourishes Lung-Yin, benefits the stomach and the heart, promotes generation of body fluids, clear heat by promoting the functions of heart, lung, stomach channels.

2. Shi Hu (Herba Dendrobii)
Shi Hu is also known as Dendrobium. The sweet, bland, slightly cold herb has been used in TCM to treat weakness after febrile diseases, thirsty, thromboangitis obliterans, chronic throat infection, blurry vision and weak lower back as it nourishes the stomach and yin, clear heat, promotes generation of body fluids by enhancing the lung and stomach channels.

c. Nu Zhen Zi (Fructus Ligustri Lucidi)
Nu Zhen Zi is also known as Glossy Privet Fruit. The bitter, sweet and neutral herb has been used to promote immune system and circulatory system, anti inflammatory, anti cancer properties, lower blood sugar and protect the liver as it tonifies the liver and kidneys, clears heat and improves vision by soothing the functions of liver and kidney channels.

d. Long Kui (Herb Solani nigri)
Long Kui is also known as wonderberry. The bitter, cold, slightly sweet and toxic herb has been used in TCM as diuretic and antipyretic and to treat acute kidney inflammation, chronic bronchitis, throat cancer, larynx Cancer, uterus cancer as it clears heat, eliminates toxin, improve urination by enhancing the function of liver, kidney and stomach channels.

e. Fu Ling (Sclerotium Poriae Cocos)
Fu Ling is also known as Indian Bread. The sweet, bland, neutral herb has been used in TCM as a diuretics and to treat insomnia or forgetfulness, loss of appetite and inflammation of the uterus and chronic sinusitis as it eliminates water, strengthens the spleen, calms the mind, clear damp-heat by enhancing the function of heart, spleen, lung, kidney channels.

f. Etc.

Foods
In traditional Chinese medicine, the herb, Ling Zhi (Reishi mushroom) is viewed to contain certain substances which process the antibacteria, antivirus and antitumor properties over thousands of year. In general public, most mushrooms contain the same values but differing in volume.
1. Shiitake or Chinese black mushroom
Shiitake contains protein, glutamic acid, low cholesterol, β-glucosidase and 18 amino acids. Some researchers found that shiitake mushroom may stimulate the immune system, possess antibacterial properties, reduce platelet aggregation, and possess antiviral properties, possibly through antiviral agents known as proteinase inhibitors. Lentinan in Shiitake enhances the immune function and prevents our body against cancer. The substance of interferon inducer may be used to treat cancer.

2. Agaricus campestris
Agaricus campestris contains high amount of B vitamin and inorganic salts. In rat study, researchers found that Agaricus campestris inhibits the progression of cancer cells.

3. Hericium erinaceus
One of the favorite food in many Chinese expensive recipe, Hericium erinaceus may inhibit the growth of tumor. In China, Some of the pharmaceutical drug company have made them in form of Mushroom slices for use in preventing and treating stomach and Esophageal cancer.

4.Tremella
Tremella contains high amount of fiber, protein, calcium, polysaccharides, etc. Researchers found that tremella enhances the immune system scavenging, T cells and B lymphocytes functions, thus preventing the progression of cancer cells.

5. Flammulina：
Beside it has been used in TCM to treat liver inflammation, ulcer and lower blood cholesterol, in 1982 study, researchers in Japan found that certain substances in flammulina help to protect our body against cells DNA mutation cause of cancer.

6. Etc.

Recipes
Coix Seed,Shepherd’s purse and pig Tripe soup
Coix Seed 100g，Shepherd’s purse 60g，pig Tripe 150 g，dried Tangerine Peel 6 g
a. Take away fat from pig tripe the cut to small slice, then clean it well with water after stirring with a little bit of salt and corn starch
b. Clean Coix seed, Sherpherd’spurse and dried tangerine peel in water
c. Put all of them in the pit with high heat, after boiling, turn to lower heat for 1-1/2 hours
The recipe is for the use to clear heat, move fluid, nourish yin and cool blood, thus treating bladder, kidney and diuretic related tumors or after surgery or chemotherapy.
** Use only for symptoms of urination with brown color blood, back and joint pain, thirsty, redden tongue, little coating.

** Not suitable for patients with liver and kidney yin deficiency, prolonged urination with blood, little color in urine, fatigue, short of breath, back and joint cold with pain, thirsty but don’t like to drink, pale yellow and smooth and tender tongue.

** Do not use this formula for patients with exogenous heat
** If you can replace pig liver with pig tripe

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is a vital organ of the human being, located near the backbone on either side of the heart with functions of inhaling oxygen from the air then transporting them to the bloodstream to nourish the body cells need and exhaling carbondioxide from the bloodstream.

is defined as a condition of the abnormal growth of the cells in the lung’s tissue. Most common form of primary lung cancers are derived from epithelial cells. In Us, Lung cancer is the leading cause of cancer deaths, causing 158,683 people deaths, including 88,329 men and 70,354 women, according to 2007 statistic.

Types of lung cancer
A. Non small cell lung cancer
Non-small cell lung cancer is the most common type of lung cancer. It usually grows and spreads methodically and predictably.Most common types of lung cancers include
1. Squamous-cell carcinoma often found in an outer area of the lung,
2. Large-cell carcinoma found in the center of the lung by an air tube
3. Adenocarcinoma found in any part of the lung.

B. Small cell lung cancer
Small cell lung cancer is a fast-growing type of lung cancer, it can spread to distant parts of the body in a relatively early stage. Most common types of small cell lung cancer include
1. Small cell carcinoma in which the cells are small, round and resemble oats.
2. Mixed small cell/large cell carcinoma in which malignant tumor found in combining of components of small cell lung carcinoma with large cell lung carcinoma.
3. Combined small cell carcinoma in which malignant tumor found in combining of a component of small cell lung carcinoma with components of non-small cell lung carcinoma.

1. Smoking
Smoking is a major cause of lung cancer as it enhances the oxidation stress as the lung inhales oxygen from the air by combining them with cancer causing chemical substances (carcinogens) in the cigarette, leading to the damage of lung’s tissue. Normally, our body can withstand and repair this damage, but prolonged exposure your lung to these danger carcinogen can result in cancer development.

2. Second hand smoke
Prolonged exposure to second hand smoke can also increase the risk of lung cancer as mentioned above.

3. Exposure to certain chemicals
There are increased risk of lung cancer for people who inhale or exposing certain industrial chemical such as radon gas, arsenic, chromium, nickel, etc. in the work place.

4. family history
Risk of lung cancer increased if one of the members of your direct family has lung cancer.

5. Lung disease
Risk of ling can caner increase if you have or previous had certain types of lung disease

6. Excessive drinking
According to the study by Freudenheim et al., R. Curtis Ellison MD suggested that alcohol intake of more than 2 drinks per day is associated with a small increased risk of lung cancer. This study, like others, suggests a weak, positive association between consuming larger amounts of alcohol (>2 drinks a day) and lung cancer risk.

7. Etc.

1. Shortness of breath.
Shortness of breath is one of most common symptoms of lung cancer, as the lung is now no longer function properly due to abnormal growth of some of its cell within it or cancer cells are growth larger enough to block the major airway, or due to fluid accumulation.

2. Coughing up blood.
Cough up blood is another symptoms of lung cancer, it can be caused by breaking off capillary due to cancer invasion or some of malignant cells. Rupture of the tumor can be extreme danger to the patient.

3. Pain
Pain also associated with the lung cancer if the cancer spread to the lung lining and suppress the nerve in the lung.

4. Fluid in the chest
Fluid in the chest may be resulted of blood or pus due to breaking of lung cancer or lung is no longer function properly in expelling mucus and phlegm, leading to shortness of breath.

5. Wheezing and hoarseness
May be due to blocking of major airway

6. Unintentional weight loss
Any unintentional weight loss over 10%

7. Bone pain
If cancer has spread to bone tissue

8. Hormones
Due to small cell lung cancer imitation of some of the body hormones, leading obesity, high blood pressure, nausea, etc.

9. Etc.

Diagnosis and tests
Family history and physical examination are always important for patient with some of above symptoms. Depending to the types of lung cancers, diagnosis and tests may include
1. Chest X ray
Chest X ray is a form of electromagnetic radiation to take image and check for any abnormality of the lung
2. CT Scan (computerized tomography)
A CT scan generates a large series of two-dimensional X-ray images taken around a single axis of rotation, to create a three-dimensional picture of the inside of the body in details.The pictures are viewed by your doctor to see the extent of the tumors abnormalities, such as spreading of cancer to the nearby structure and lymph nodes. CT scan can only review the existence of cancer . but can not tell it is a primary or secondary cancer.

3. MRI (magnetic resonance imaging)
MRI (magnetic resonance imaging) is one of many advanced technology used to visualize internal structures cross sectional imaging of your body used effectively in providing the better details of the metastasis of cancer in the lung and surrounding areas.

4. Sputum Cytology
Sputum Cytology is a test used to examine a sample of mucus under a microscope to check for any abnormal cell.

5. Bronchoscopy
Bronchoscopy is a procedure used to look inside the airways of the lung by inserting a thin, flexible tube called a bronchoscope with a light and small camera that allow your doctor to see the airways and take pictures to check for any abnormality in the trachea and airways, such as infection, inflammation, or malignant tumor, through nose or mouth, down into the airways.

6. Lung Biopsy
In lung biopsy, a sample of liver is taken by a thin, tube-like instrument to examine under microscopy to review the stage of the cancer.

7. Bone scan
With a small amount of radioactive substance, called a tracer injected into a vein, as it travels through the bloodstream it allows a special camera takes pictures of the tracer in your bones. If there is any abnormality in the bone, it will show up in the pictures.

8. Mediastinoscopy
Mediastinoscopy is a surgical procedure to examine the inside of the upper chest between and in front of the lung by inserting of a mediastinoscope through the opening made in the neck just above the breastbone to collect sample to test under microscope for signs of infection, inflammation, or malignant tumor.

9. Positron emission tomography (PET scan)
Positron emission tomography (PET scan) is a type of nuclear medicine imaging with the uses of small amounts of short-lived radioactive material, either injected into a vein, swallowed or inhaled as a gas which will appear in the area of the body being examined, where it gives off energy in the form of gamma rays detected by a camera of positron emission tomography that produces a three-dimensional image or picture of functional processes of the organ in the body.

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11. Etc.

The Grade of lung cancer is depending to the tendency of spreading. Low grade cancers usually grow more slowly and are less likely to spread while high grade cancer indicates otherwise.

1. Stage 0
Lung caner is classified as Stage 0, if the cancerous cell have not penetrated in deeper tissue but in the surface of the lung lining.
2. Stage I
In stage I, The cancerous cells are no longer in the surface but have invaded into deep lung lining, but still completely inside the lung.
a. Stage IA1
The cancer is ≤ 3 mm (1/8 inch) deep and ≤ 7 mm (1/4 inch) wide.
a.1. Stage IA1: The spreading is < 3mm(1/8 inch) deep and & less than 7mm (1/4 inch) wide.
a.2. Stage IA2: The invasion area is ≥ 3 mm but ≤ 5 mm (about 1/5 inch) deep and & less than 7 mm (about 1/4 inch) wide.
b. Stage IB: The cancer in this stage have invaded the connective tissue, & less than 5mm (1/5 inch).
b.1. Stage IB1: Cancer is 4 cm large (1 3/4 inches).
b.2. Stage IB2: Cancer is ≥ 4 cm (1 3/4 inches) but & less than 5cm (1/5 inch)

3. Stage II
In stage II, the cancerous cells have spread to distant tissues, but is still within the lung.

4. Stage III
n this stage, cancerous cells has spread to the tissues immediately surrounding the lung.

5. Stage IV
In stage IV is the most advance stage of lung cancer as cancerous cells have spread to the distant parts of the body
In this stage, cancerous cells has spread to the tissues immediately surrounding the lung.

Prevention
A. How to avoid
1. Quit smoking
According to statistic, approximate 87% of lung cancer deaths are caused by smoking.
2. Eating more fruits and vegetables
This foods contain high amount of antioxidants which enhance the immune system against the forming of free radicals and prevent the alternation of cell DNA cause of abnormal cell growth.
3. Second smokes
Second smoke contains same amount of carcinogens, exposure to it can increase the risk of lung caner
4. Environment chemicals
avoid exposure to certain industrial chemical such as radon gas, arsenic, chromium, nickel, etc.. In the work place, deal with them by taking maximum pre-cautions and prevention.

5. Alcohol
Researcher found that increased risk of lung cancer for peopne who have >2 drinks a day.

6. Etc,

B. With diet
Traditional Chinese medicine link all foods with white color to lung tonification, taking them are associated reduced risk of lung cancer.
1. Cruciferous Vegetables
Cruciferous vegetables such as cauliflower, broccoli, cabbage, etc. beside contain high amount of antioxidants but also in a study of AACR FCPR 2008: Cruciferous Vegetables Reduce Lung Cancer Risk in Smokers, reseachers found that there is a significant inverse association between consumption of cruciferous vegetables and lung cancer was only seen for squamous or small cell carcinoma, which are the 2 subtypes most strongly associated with heavy smoking.

2. Pomegranate juice
In a recent issue of Cancer Research, researchers led by Hasan Mukhtar, co-leader of the Cancer Chemoprevention Program of the University of Wisconsin Paul P. Carbone Comprehensive Cancer Center, demonstrate that drinking pomegranate fruit extract helps slow the growth of lung cancer in mice found that pomegranate fruit continues to show great promise,” says Mukhtar, professor of dermatology at the School of Medicine and Public Health and a member of the Carbone Cancer Center. “We have earlier shown that pomegranate fruit contains very powerful skin and prostate cancer-fighting agents. These recent findings expand the possible health benefits of the fruit to the leading cause of cancer death in the country and worldwide: lung cancer.”

3. Turmeric
In Molecular Nutritional and Food Research, March 2008, researchers reported that naturally occurring polyphenols in curcumin offer a safer alternative treatment. Curcumin can directly scavenge free radicals such as superoxide anion and nitric oxide, and modulate important signaling pathways. These polyphenols also down-regulate expression of pro-inflammatory mediators, and up-regulate desirable gene expression in the lungs. Researchers concluded that curcumin is a potential therapeutic agent against chronic lung diseases.

4. Apple
In the Article: “Triterpenoids isolated from Apple Peels Have Potent Antiproliferative Activity and May be Partially Responsible for Apple’s Anticancer Activity”, Rui Hai Liu and Xiangjiu He pointed out that apple consumption has been linked to a reduced risk of chronic health problems such as lung cancer, heart disease, and stroke.

5. Garlic
An analysis of several case-controlled studies in Europe suggests an inverse association between garlic consumption and risk of common cancers.

6. Etc.

C. With nutritional supplements

1. Antioxidants
Antioxidants such free radical scavengers vitamin A, C, E enhance the immune system against the forming of free radicals and prevent the alternation of cell DNA cause of abnormal cell growth.

2. Quercetin
Quercetin is a type of phytochemical, also known as a flavonoid. In cell culture or animal studies, researchers found that quercetin has activity against some types of cancer cells and suggested it may be potential benefits in treating cancer.

3. Naringin
In s study of Intake of Flavonoids and Lung Cancer, researcger found that flavonoids, such as quercetin and naringenin (the aglycone derived from naringin), inhibit certain cytochrome P450 enzymes (CYP1A1 and CYP3A4, respectively) involved in the bioactivation of chemical carcinogens.

4. Selenium
In a study of selenium effects in skin cancer, researchers discovered that it might help prevent lung cancer instead according to the book of 1001 Home Health Secrets for Seniors.

5. Lycopene and 1,25-dihydroxyvitamin D3
In a study of Lycopene and 1,25-dihydroxyvitamin D3 cooperate in the inhibition of cell cycle progression and induction of differentiation in HL-60 leukemic cells, researchers found that The combination of low concentrations of lycopene with 1,25-dihydroxyvitamin D3 exhibited a synergistic effect on cell proliferation and differentiation and an additive effect on cell cycle progression. Such synergistic antiproliferative and differentiating effects of lycopene and other compounds found in the diet and in plasma may suggest the inclusion of the carotenoid in the diet as a cancer-preventive measure.

6. Etc.

Treatments
A. In conventional medicine
A.1. Non small cell lung cancer
1. Surgery
a. The aim of the treatment is to cure the cancer with surgery, if possible. Otherwise, treatments are focused to treat and control the symptoms as long as possible. If the cancer is located in the outer parts of the lung and has not spread to the lymph nodes, then surgery will be effective in curing it.
b. Risks and side effects
b.1. Surgical and anesthesia risks
b.2. Shortness of breath
Due to remove parts of the lung
b. 3. Etc.

2. Radiotherapy
By using high-energy x-rays or other types of radiation, radiation therapy kills lung cancer cells and keep them from growing or regrowing. Depending to stage or grade there are two types of radiation therapy.
a. External radiation
By using a machine outside the body to send direct high-energy x-rays or other types of radiation toward the cancer.
b. Internal radiation
By placing a radioactive substance direct into or near the cancer by a medical instrument with the aim to kill nearby cancer cells.
c. Side effects
c.1.. Fatigue
c.2. Chest pain
c.3. Heart problem
c.4. Short of breath
c.5. Skin discoloration or pinkness, irritation.
c.6. Etc.

3. Chemotherapy
a. Chemotherapy is most use to treat with advance stage of lung cancer, as it has spread to a distant parts of the body by using drugs taken by mouth or injected into a vein or muscle of the patient to stop the growth of or to kill cancer cells.
b. Side effects
b.1. Nausea
b.2. Vomiting
b.3. Hair loss
b.4. Fatigue
b.5. Anemia
b.6. Mouth sores taste and smell changes
b.7. Infection
b.8. Etc.

4. Biological therapy
a. Biological therapy with the use of Iressa, Taceva, target binding agents have contributed significantly in treating non small cell lung cancer by binding to the cancerous site and preventing its activity.
b. Risks and side effects
b.1. Weight gain
b.2. Neutropenia
b.3. Headache,
b.4. Thrombocytopenia
b.5. Anemia
b.6. Nausea
b.7. Rash
b.8. Etc.

A.2. Small cell lung cancer
Small cell ling cancer is behave differently than non small cell lung cancer as it is invasive and has tendency to spread to distant parts of the body, however, it often responds well to the treatments of chemo and radio therapies
1. Chemotherapy
a. Chemotherapy is most use to treat small cell lung cancer, as it has spread to a distant parts of the body by using drugs taken by mouth or injected into a vein or muscle of the patient to stop the growth of or to kill cancer cells.
b. Side effects
b.1. Nausea
b.2. Vomiting
b.3. Hair loss
b.4. Fatigue
b.5. Anemia
b.6. Mouth sores taste and smell changes
b.7. Infection
b.8. Etc.

2. Radiotherapy
By using high-energy x-rays or other types of radiation, radiation therapy kills lung cancer cells and keep them from growing or regrowing. Depending to stage or grade there are two types of radiation therapy.
a. External radiation
By using a machine outside the body to send direct high-energy x-rays or other types of radiation toward the cancer.
b. Internal radiation
By placing a radioactive substance direct into or near the cancer by a medical instrument with the aim to kill nearby cancer cells.
c. Side effects
c.1.. Fatigue
c.2. Chest pain
c.3. Heart problem
c.4. Short of breath
c.5. Skin discoloration or pinkness, irritation.
c.6. Etc.

3. Surgery
It is not very helpful to remove the primary lung tumor because of its spreading tendency, but it can be used to reduce certain symptoms such as enlarged lymph nodes, spleen, etc.

4. Etc.

B. Herbal medicine
1. Grape Seed
In a studies of grape seed extracts in test tubes, researchers suggested that it may prevent the growth of breast, stomach, colon, prostate, and lung cancer cells.

2. Green tea
In some clinical studies researchers suggested that the polyphenols in green tea, may play an important role in the prevention of cancer by killing cancerous cells and stoppimg their progression.

3. Aloe
in some studies, researchers suggest that some chemical compounds of aloe, such as acemannan, aloeride, and di(2-ethylhexyl)phthalate (DEHP) may have immunomodulating and anticancer effects.

4. Cat’s claw
In vitro studies, researchers found that cat’s claw demonstrated anticancer effects against several cancer cell lines.

5. Etc.

C. Traditional Chinese medicine
1. Huang Qi
In study of Astragalus-Based Chinese Herbs and Platinum-Based Chemotherapy for Advanced Non–Small-Cell Lung Cancer: Meta-Analysis of Randomized Trials, researchers found that astragalus may increase effectiveness of platinum-based chemotherapy when combined with chemotherapy. These results require confirmation with rigorously controlled trials.

2. Yun Zhi
In a study by by Dr. Kenneth Tsang at the University of Hong Kong’s School of Medicine in 1999, on the polysaccharopeptide (PSP) treatment of patients with advanced non-small cell lung cancer, researchers found that after the four-week treatment, there was a significant increase in blood leckocyte and neutrophil levels and body fat compared with pre and post treatment of PSP. Serum IgG and IgM were significantly improved in the PSP treated group compared to the placebo group after four weeks In addition, there were less PSP treated patients who withdrew from the study due to disease progression. Therefore, this study suggests that PSP treatment may be of some benefit in patients with NSCLC.

3. Ling Zhi
In an study of Ling Zhi extracts tested them on three different types of cells: drug-sensitive small-cell lung cancer, drug-resistant small-cell lung cancer, and normal lung cells, researchers found that Ling Zhi killed lung cancer cells. Cancer cells responded to the herb much in the same way as they would react to chemotherapy drugs. Yet unlike chemotherapy drugs, which can also be toxic to healthy cells, herbal extracts were more deadly to cancer cells than to normal cells, indicating that they have some ability to specifically target cancer.

4. Yi Yi Ren
About 20 years ago, a young physician named Dr. Da-Peng Li observed that people in China who ate coix seed as a dietary staple had a surprisingly low incidence of cancer. Research in test tubes, animals and humans by Zhejiang Kanglaite Pharmaceutical Company indicated that The drug from the seed’s oil, Kanglaite has been shown to induce apoptosis, or programmed cell death, in various kinds of human tumors, and has a powerful effect on angiogenesis (the formation of new blood vessels to feed the tumor).

5. Etc.

For other health articles, please visit http://medicaladvisorjournals.blogspot.com/

Please follow me at http://twitter.com/kylejnorton